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A different gene near the hairless gene on chromosome 8p21 causes a rare hair loss condition in a German family.

A protein called HMGB1 helps hair grow by affecting prostaglandin metabolism.

ME1 and PPAR signaling may influence hair loss in androgenetic alopecia.

BMP4 helps stem cells turn into pigment-producing cells, affecting hair color and growth.

Plasmalogens activate a channel in cells that may stimulate hair growth.

Balding hair follicles have reduced growth factors and increased inhibitory factors, suggesting new treatment paths for hair loss.

A gene variant causes patched hair loss in mice, similar to alopecia areata in humans.

Proteomic analysis can identify genetic differences in mouse hair, helping understand hair defects and variations.

The research suggests that immune cells and a specific type of cell death called ferroptosis are involved in Frontal fibrosis alopecia.

Certain immune cells in the skin release a protein that stops hair growth by keeping hair stem cells inactive.

Mutations in the LIPH gene cause woolly hair in a child.

CD8A and FOXD2-AS1 may be key for diagnosing and treating alopecia areata.

COX2 and ATP synthase control the size of hedgehog spines.

Krt6a-Cre transgenic mice help study gene effects on hair follicle development and tumor suppression.

Keratin 17 is linked to various diseases, including cancer and skin conditions, and may be a target for diagnosis and treatment.

The research identified unique metabolic activities in immune cells associated with hair loss in Alopecia Areata.

Mycophenolic acid may help treat hair loss by promoting hair growth and cell proliferation.

Researchers found a gene in hamsters that responds to male hormones and may be indirectly controlled by them.

Restoring hair follicle immune privilege may help treat alopecia areata.

PROTACs show promise in cancer treatment by effectively degrading specific harmful proteins.

Frontal fibrosing alopecia shows increased inflammation and JAK-STAT pathway activity without reduced hair proteins.

Alpha 6 + /MHCI - cells have stem cell traits and are similar to mouse hair follicle stem cells.

Different body parts have varying levels of certain hair follicle markers.

Malondialdehyde-modified DNA may trigger an immune response in alopecia areata patients.

Wnt signaling controls whether hair follicle stem cells stay inactive or regenerate hair.

The enzymes Tet1, Tet2, and Tet3 are important for the development of hair follicles and determining hair shape by controlling hair keratin genes.

Autophagy is important for determining the protein makeup of hair.

A specific type II hair keratin was identified and found in hair cortex and tongue cells.

High ornithine decarboxylase levels may lead to hair loss and cancer by increasing CK2 activity in the nucleus.