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A new mouse gene, Keratin 17n, is mainly found in nail tissue and may explain why mice without Keratin 17 don't have nail issues.

AP-1 controls tumor cell type by affecting key signaling pathways.

The gene NM_026333 slows down aging by affecting the NCX1 pathway and could be targeted for anti-aging treatments.

LRIG1 is linked to better survival in Merkel cell carcinoma.

Personalized sonidegib dosing can effectively treat Gorlin-Goltz syndrome with fewer side effects.

PBX1 helps hair stem cells grow and change by turning on certain cell signals and preventing cell death, which may be useful for hair regrowth treatments.

The FTO gene hinders stem cells in hair follicles from becoming pigment cells.

Polycomb Repressive Complex 2 is not needed for hair regeneration.

A mother's PPARγ is crucial for preventing harmful milk that can cause inflammation and growth problems in babies.

Protein kinase C epsilon may promote skin cancer development after UV exposure by affecting nearby cells.

BMP signaling affects hair color by interacting with the MC-1R pathway.

A genetic duplication on chromosome 5 was linked to a woman's unique combination of medical conditions.

A mouse gene mutation increases the risk of skin cancer.

The protein CCN2 controls hair growth by affecting hair follicle formation and stem cell activity in mice.

The peach gene pCTG134 helps control the interaction between auxin and ethylene hormones during fruit ripening.

The mutation causes hairless mice due to mislocalized and dysfunctional HR protein.

Protein kinase C epsilon may increase skin cancer risk by affecting nearby cells.

Higher activity in lichen planopilaris is linked to certain immune and tissue genes.

A mutation in the Adam10 gene causes freckle-like spots on Hairless mice.

Too much parathyroid hormone-related protein in skin disrupts hair growth in mice.

Mice can correct hair follicle orientation without certain genes, but proper overall alignment needs those genes.

Researchers found key regions in the mouse hairless gene that control its activity in skin and brain cells, affecting hair follicle function.

CXXC5 can both suppress and promote cancer, making it a complex target for treatment.

A gene mutation in mice causes hair loss, weak bones, and protein buildup, showing how protein processing issues can lead to diseases.

The ACTH/MC2R system is crucial for controlling hair growth cycles in mice.

A new mouse mutation causes skin and hair issues, influenced by another gene.

c-Maf positive macrophages help form hypertrophic scars by affecting fibroblasts and collagen production.

GPRC5D is linked to the formation of hair, nails, and certain tongue areas.

A new mutation in the TRPS1 gene caused Trichorhinophalangeal syndrome in a 17-year-old, highlighting the need for genetic testing.

A specific gene mutation is linked to a hereditary form of dystonia that responds well to certain medications.