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Carriers of a specific gene mutation have subtle skin changes without visible symptoms.

Modified β-catenin can cause different effects in mouse skin cells, leading to cysts or tumors depending on the cell type.

KRT17 may be a new target for endometrial cancer treatment because it helps cancer cells move and form new blood vessels.

Blocking YAP/TAZ could be a new way to treat skin cancer.

Activating cAMP and ATP improves hair growth and strength.

The study found that variations in hair protein genes are likely due to evolutionary deletions or duplications.

Natural compounds may help treat advanced papillary thyroid cancer by targeting specific molecular pathways.

Mice with human skin protein K8 had more skin problems and cancer.

Annexin A2 isoform 2 helps dermal papillae cells grow, affecting hair growth.

Keratins K6 and K16 are expressed more freely in regenerating mouse skin than K1 and K10.

Mutant MK6a transgenes in mice cause blistering, hair loss, and potential human alopecia.

New RIPK4 gene mutations were found to cause a type of skin and limb birth defect.

Nilotinib can cause skin issues like red bumps and hair loss.

Cancer cells often have more copies of TERT and TERC genes, which helps them grow and could affect patient outcomes.

Patched1 helps prevent tumors by controlling cell growth.

A KLK5 inhibitor effectively improved skin symptoms in a mouse model of Netherton Syndrome.

Krtap11-1 is important for hair strength and structure.

Tet2 and Tet3 enzymes are essential for controlling hair growth by affecting DNA demethylation and gene expression in mice.

Skin tumor cells in patients with tuberous sclerosis have higher levels of a protein called cathepsin B.

Inhibiting TYK2 can restore hair growth in alopecia areata.

Transit-amplifying cells are crucial for tissue repair and can contribute to cancer when they malfunction.

New biological pathways and potential treatment targets for male pattern baldness were identified.

Deleting podoplanin in mice promotes hair growth by enhancing cell migration.

Cyclosporin A, a drug, reduces TGF-β2 expression in skin cells, potentially causing excessive hair growth through a process involving the calcineurin/NFAT pathway.

The TCL1 transgenic mouse model is useful for understanding human B-cell leukemia and testing new treatments.

Keratin 10 end domains may increase skin cancer risk by reducing cell death.

A gene variant causes a skin and hair disorder by disrupting protein balance.

The DMI3 gene is essential for nodule development and symbiosis in certain plants.

A specific gene change in APCDD1 increases the risk of hair loss.

A20 protein is crucial for normal skin and hair development.