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A gene deletion in mice causes weak protein, immune issues, hair loss, airway problems, and wasting disease.

Overexpressing PKCepsilon in mice reduces papillomas but increases carcinomas.

The humanized AA mouse model is better for testing new alopecia areata treatments.

PINK1 is important for controlling gut immune responses linked to early Parkinson's disease.

The African spiny mouse can fully regenerate its muscle without scarring, unlike the common house mouse.

Mutations in claudin-1 and claudin-3 cause hair loss in baby mice.

The research found genetic differences in identical twins that could explain why one twin has a disease while the other does not.

Vitamin D receptor helps prevent skin tumors.

Inhibiting mTORC2 can reduce DNA repair and increase cancer cell death, suggesting potential for targeted brain cancer treatments.

People with PCNT mutations often develop severe insulin resistance and early-onset diabetes during childhood or adolescence.

New mutations in the hairless gene may cause hair loss and affect bone development.

New mutations in hair keratin genes can change hair structure and cause monilethrix, with nail issues more common in certain gene mutations.

CXCL7 is essential for muscle repair by aiding early neutrophil infiltration.

Mutations at Val-889 and Arg-752 disrupt key interactions in the androgen receptor, affecting its function.

Researchers found three types of melanocytes in developing mouse skin, each with different genes and locations.

A mutation in the KRTHB5 gene causes hair and nail issues.

New mutations in the DSG4 gene cause a rare hair condition.

Budesonide and N-acetylcysteine reduced tumors and alopecia in mice, regardless of FHIT gene status.

New mutations in the VDR gene cause vitamin D-resistant rickets without hair loss.

Chronic T. gondii infection may harm male fertility.

A bull with a gene mutation was asymptomatic, synthetic retinoids cause hair loss, and new therapeutic targets were identified for skin diseases.

Identical twins with a rare KRT 86 gene mutation both have the hair disorder monilethrix.

Knocking out certain genes in mice helps understand skin and hair growth problems.

Basal cell carcinomas in mice can start from hair follicle stem cells and other skin cell types, depending on signaling levels.

Some cells may slow melanoma growth, a protein could affect skin pigmentation, a gene-silencing method might treat hair defects, skin bacteria changes likely result from eczema, and a defensin protein could help treat multiple sclerosis.

Mice without certain skin enzymes have faster hair growth and bigger eye glands.

The 1298CC genotype in RA patients leads to higher methotrexate toxicity, while the 1298AA genotype results in better treatment response.

Mice with human fetal thymic tissue and stem cells developed symptoms similar to chronic graft-versus-host disease.

The enzymes Tet1, Tet2, and Tet3 are important for the development of hair follicles and determining hair shape by controlling hair keratin genes.