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Researchers found a new gene variant linked to a rare bone disease, which doesn't always cause symptoms in carriers.

Melanocyte pathology requires keratinocyte hyperplasia and regulation dysfunction.

The MTHFR C677T mutation may increase the risk of alopecia areata in the Turkish population.

The TRPV3 gene mutation affects hair growth by keeping mice in the growth phase longer, which could help treat hair loss.

Understanding genetic variations in mice is crucial for studying skin, hair, or nail abnormalities.

External factors can cause skin cancer cells that usually don't spread to grow and form tumors in mice.

Scientists made a mouse that can be made to lose hair and then grow it back.

A mutation in mice causes hair loss and immune problems.

Mouse Scube3 affects teeth, tongue, vibrissae, and eye development, but not facial structure or limb growth.

Msx2-deficient mice experience irregular hair growth and loss due to disrupted hair cycle phases.

A gene mutation worsens skin irritation in mice due to a lack of certain fats.

A specific metabolite, not a receptor protein, triggers the production of certain kidney enzymes, but this process is disrupted in mice with a mutation causing testicular feminization.

MC-DNA vector-based gene therapy can temporarily treat CBS deficiency in mice.

Mice with human-like EGFR had growth issues, skin defects, heart problems, and unusual bone development.

Mutant MK6a transgenes in mice cause blistering, hair loss, and potential human alopecia.

A mouse gene mutation increases the risk of skin cancer.

Hair loss in certain mice is linked to changes in keratin-related genes.

Putting thymidine dinucleotide on newborn mice's skin can delay and reduce skin cancer.

Deleting TNFα gene reduces skin cancer risk in certain mice.

Cathepsin L deficiency causes hair and skin issues in mice.

Mutations in the hHb6 gene cause the hair disorder monilethrix.

New mouse models help study melanocytic cells for melanoma research.

A specific gene mutation is linked to a hereditary form of dystonia that responds well to certain medications.

Loss of Ten1 in mice causes telomere shortening and symptoms similar to human dyskeratosis congenita.

A new mutation in the KRT86 gene causes a hair disorder with variable expression.

The mouse model showed defects in adult stem cell maintenance related to Hutchinson-Gilford progeria syndrome.

Overexpressing the mineralocorticoid receptor in mouse skin causes skin thinning, early skin barrier development, eye issues, and hair loss.

Mice without the vitamin D receptor are more prone to UV-induced skin tumors.

Modified β-catenin can cause different effects in mouse skin cells, leading to cysts or tumors depending on the cell type.