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AGD1 is important for root hair development in Arabidopsis, working with phosphoinositide signaling and the actin cytoskeleton.

A new gene mutation causes vitamin D-resistant rickets and hair loss in two siblings.

A20 protein is crucial for normal skin and hair development.

Noggin overexpression delays eyelid opening by affecting cell death and skin cell development.

A deletion in the CDH3 gene causes a rare disorder with short hair and vision loss.

Stopping the beta-blocker nadolol led to quick hair regrowth and improved scalp condition.

Niacin derivatives may increase hair fullness in women with hair loss.

Different enzymes are active in different parts of developing mouse organs.

A 10-year-old boy's nail condition is mainly cosmetic and improves over time with treatment.

N6-methyladenosine affects hair follicle development differently in Rex and Hycole rabbits.

Antisense oligonucleotides show promise for treating Myotonic Dystrophy type I.

NADPH oxidase and phospholipase D help root hairs grow by activating calcium channels.

PAD enzymes play a key role in hair growth and structure.

Nicastrin deficiency may cause skin cell damage and pigmentation disorders in humans, similar to effects seen in fish.

A woman with dermatomyositis improved after treatment for neck panniculitis, suggesting similar treatment for related conditions.

Farudodstat may effectively treat alopecia areata by protecting hair follicles without harmful effects.

Scientists created tiny particles that release medicine on the skin and in hair, working better at certain pH levels and being safe for skin cells.

Newly made nicotinamide compounds could potentially treat cancer.

DcR3 helps heal wounds and regrow hair by changing macrophages to a repair-focused type.

PEDF reduces oxidative damage and supports stem cells.

HOXC13 is essential for hair and nail development by regulating Foxn1.

Low-dose naltrexone and platelet-rich plasma can regrow hair in lichen planopilaris.

NuMA-microtubule interactions are vital for proper skin structure formation and function.

Neurotrophins regulate nerve growth by balancing promotion and suppression.

Ectodermal dysplasia syndromes are caused by disruptions in key signaling pathways affecting tooth and hair development.

JMJD3 and NF-κB activate Notch1, which is essential for skin cell movement and wound healing.

A new compound effectively inhibits human 5α-reductase 1.

Tofacitinib effectively improved severe skin symptoms in a patient with Hypohidrotic Ectodermal Dysplasia.

The new model helps understand and develop treatments for genetic skin disorders like AEC.

Boys with less severe EDA mutations in XLHED have milder symptoms and better sweat and hair production.