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Progesterone and its byproducts control a specific receptor in the brain independently of progesterone receptors, affecting conditions related to the menstrual cycle.

Early-life neurosteroid changes affect adolescent exploration and adult behavior.

Neurosteroids from glia cells help control seizure development in epilepsy.

Sex and stress steroids quickly change brain cell structures in the hippocampus.

Progesterone and allopregnanolone increase glycine release in rat brain cells.

Midazolam impairs learning and memory by increasing neurosteroids through specific receptor activation.

The tested hair dye ingredients do not form harmful oxidized metabolites in the liver.

Neonatal allopregnanolone and stress affect behavior differently in adolescence and adulthood.

5α-reduced neurosteroids may help regulate glial cell differentiation.

The availability of certain hormones and specific stimulation patterns affect long-term synaptic changes in the male rat brain.

Progesterone and testosterone protect brain cells from damage through specific pathways.

TTNPB helps turn stem cells into neural stem cells, improving depression-like behaviors in rats.

Neurosteroids in the brain can increase or decrease seizure risk in mice.

Drugs localize differently in hair strands based on their properties.

Certain substances can help skin cells become anti-inflammatory, aiding in tissue repair.

Blocking neurosteroid production can lead to more seizures and faster epilepsy onset in rats.

Male tissue has more cell death than female tissue after ischemia, and some neurosteroids only protect female cells.

Human dermal fibroblast proteins help restore nerves during healing.

Pregnancy and nursing increase certain brain activities in rats, but these changes disappear when the babies are taken away.

Morphine increases testosterone breakdown in the rat brain and spinal cord.

Neonatal neurosteroid levels affect adult brain function and behavior.

Testosterone metabolites may help relieve diabetic nerve pain.

Progesterone increases alcohol tolerance and withdrawal anxiety, while DHEAS prevents them.

Paroxetine relieves pain by increasing allopregnanolone levels.

Blocking CYP17A1 enzyme may help improve certain brain function issues related to dopamine.

Tiny particles called anionic squarticles can effectively remove a common antidepressant from the body after an overdose.

Two non-steroidal antiandrogens, RU 58841 and RU 56187, form a common metabolite at different rates, which may influence their effects; RU 56187 could be used for prostate cancer treatment and RU 58841 for acne treatment.

Psychotropic drugs can interact with natural products, affecting their effectiveness and safety.