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Neuronatin is found in specific cells within rat testis, hair follicles, tongue, and pancreas, suggesting it has various roles in tissue development and function.

Hormones and neuroendocrine factors control hair growth and color, and more research could lead to new hair treatment options.

A new method improves protein analysis in hair, aiding health and disease research.

PIEZO1 helps keep hair follicle stem cells inactive, affecting hair growth.

Mutations in the FOXN1 gene cause severe immune issues but don't affect hair and nails.

Hair follicles are hormone-sensitive and involved in growth and other functions, with potential for new treatments, but more research is needed.

TTNPB helps turn stem cells into neural stem cells, improving depression-like behaviors in rats.

Androgens help motor neurons grow by increasing neuritin.

Rat vibrissae have sensory terminals with specific structures that help detect hair movements.

Glycine likely affects dendrites connected to hair follicle terminals in rats.

Ca²⁺-mediated protein citrullination controls cell growth in the CNS and may help treat brain tumors.

Phosphorylation controls TFEB's location in the cell, affecting cell metabolism and stress response.

The mutation causes hairless mice due to mislocalized and dysfunctional HR protein.

A special receptor in sensory nerve endings helps control how they respond to stretching.

Laminin-511 is crucial for early hair growth and maintaining important hair development signals.

HFMs can help study hair growth and test potential hair growth drugs.

TGF-β2 helps yak hair follicles enter the regression phase, while HSP70 tries to prevent it.

Mouse touch-sensitive nerve cells adjust their connections based on competition with other similar cells.

Mouse high-glycine/tyrosine proteins have distinct patterns in hair follicles, peaking at specific hair cycle days.

Hair follicle stem cells could help repair nerves and avoid ethical issues linked to embryonic stem cells.

Defective nuclear transport may cause gene expression changes in Progeria.

The study improved understanding of keratin fiber structure by showing consistent microfibril diameter but varying distances and electron density profiles.

Mutations in the KRT85 gene cause hair and nail problems.

Mechanoreceptors convert physical touch into electrical signals through specialized nerve structures.

The hair defect is due to abnormal inner root sheath keratinization.