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The transcription factor Meis2 is essential for touch sensation and proper nerve development in touch receptors.

A new neural network helps identify key regulators in cell changes, aiding in understanding diseases and finding new treatments.

Blocking autophagy worsens lipid buildup and dysfunction in brain cells after injury.

Fibroblasts can be mistaken for neural cells, so functional validation is needed.

Different body areas have unique skin cell communication patterns, explaining why certain skin diseases occur in specific regions.

Nerves and chemicals in the body can affect hair growth and loss.

Targeting specific biomarkers and immune signals is more effective and safer for treating inflammatory skin diseases.

miR-214-3p helps nerve repair and recovery.

Monocytes in hair follicles help neutrophils move through the skin in contact dermatitis.

Stress can affect skin and hair health through hormones.

Wnt-signaling is regulated differently in skin cells and immune responses during wound healing.

Hair follicle and adipose cell vesicles both protect neurons and reduce inflammation similarly.

Hormones during pregnancy and lactation keep skin stem cells inactive, preventing hair growth.

Stem cell activity influences autoimmune disease outcomes by affecting immune responses and tissue regeneration.

GDNF signaling helps in hair growth and skin healing after a wound.

Hair follicles on the scalp interact with and respond to the nervous system, influencing their own behavior and growth.

The WNT signaling pathway is crucial for mesenchymal stem cells' function and therapy success.

Stem cells can move to brain injury sites and be tracked, showing promise for treating brain diseases.

Microglia, the brain's immune cells, may contribute to Polycystic Ovary Syndrome (PCOS) by altering the female brain's structure and function, with kisspeptin neurons and GABA neurotransmitters also playing a role.

Stress and emotional factors can worsen skin conditions by affecting the immune system.

New insights into cell communication in psoriasis suggest innovative drug treatments.

Nucleic acids trigger chemokine production in skin cells, affecting skin inflammation.

Biomimetic nanovesicles can speed up diabetic wound healing by regulating immune cell behavior and metabolism.

Higher levels of certain nerve-related proteins are linked to hair loss in women with scalp pain.

β-Catenin signaling is involved in brain cell growth after injury and could be a therapy target.

Endogenous neurosteroids may set a baseline mood.

Progesterone initially worsens but later reduces neuropathic pain in mice, through different mechanisms.

RNase L hinders hair follicle regeneration by altering immune signals.