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Acute stress increases Y1 receptor gene expression in certain brain areas, but repeated stress does not.

Dihydrotestosterone increases certain inflammatory signals in skin cells, potentially contributing to acne.

Stress worsens hair loss in androgenetic alopecia.

A thorough, team-based approach and clear communication improve outcomes in complex neuropsychiatric lupus cases.

Type I interferons play a key role in the development of various skin diseases.

IL-17 plays a key role in severe hair loss in chronic alopecia areata.

Cerebronal® may help improve certain brain health markers.

Botulinum toxin A helped stop hair loss and grow new hair in mice.

Managing stress and nerve activity can help treat segmental vitiligo and white hair.

Macrophages and fibroblasts help repair organs after injury, affecting whether they regenerate or scar.

Stress may contribute to hair loss in alopecia areata by affecting immune responses and cell death in hair follicles.

Sphingosine 1-phosphate helps control mechanical pain.

Acne patients' skin, both with and without lesions, shows a strong immune response and higher antimicrobial activity.

New research suggests treating hair loss should focus on common inflammation rather than individual molecules.

A specific genetic change is linked to mental disorders, intellectual disability, and possibly autoimmune disease in a family.

Neuroendocrine paraneoplastic syndromes often show skin changes, helping early diagnosis and requiring a team approach for best care.

Blocking neurosteroid production can lead to more seizures and faster epilepsy onset in rats.

Pregnancy and nursing increase certain brain activities in rats, but these changes disappear when the babies are taken away.

Cannabidivarin (CBDV) helps new brain cells grow and develop in a specific brain area through a certain receptor.

Inorganic nanomaterials can improve brain disease imaging by being more precise and faster than traditional methods.

Loss of OGG1 increases skin inflammation and auto-antibodies in lupus.

TNFα and interleukin-1 blockers reduce skin inflammation from EGFR antibody therapy.

TNFα and interleukin-1 blockers reduce skin inflammation from EGFR antibody therapy.

Extracellular vesicles could offer precise treatments for psychiatric conditions by targeting brain networks.

TEC kinases may help cause inflammation in vitiligo and could be targeted for treatment.

Traumatic panniculitis can cause increased hair growth in affected areas.

Blocking COX, especially COX-2, in the skin can reduce inflammation and pain and may help prevent skin cancer.

A woman with dermatomyositis improved after treatment for neck panniculitis, suggesting similar treatment for related conditions.