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Hair color production in mice is closely linked to the hair growth phase and may also influence hair growth itself.

LPA3 signaling in the uterus is crucial for placental formation and fetal development.

The authors confirmed that β-estradiol inhibits hair growth in mice when used at the correct concentration.

Steroid hormones change the size, charge, and stability of DPPC liposomes.

The EDA pathway plays a key role in bone development by interacting with other signaling pathways.

Male hormones cause different growth in identical human hair follicles due to their unique epigenetic characteristics.

The pituitary gland functions normally even after losing most SOX2+ stem cells.

New steroidal compounds moderately block an enzyme related to testosterone conversion, less effectively than finasteride.

The L457(3.43)R mutation in the human lutropin receptor causes increased activity and hormone insensitivity, leading to precocious puberty.

Four transcription factors can convert mouse cells into hair cell-like cells, aiding hearing loss research and treatment.

Deleting the PTEN gene in mice causes nerve cells to grow larger and heal better after injury, but may cause overgrowth and hair loss in older mice.

Cortexolone 17a-propionate may be an effective new treatment for hair loss.

MicroRNA-200 prevents sebaceous gland development by inhibiting SOX9 and cell cycle progression.

Vitamin D and glucocorticoids affect bone growth, metabolism, and immune response.

Dexamethasone reduces inflammation and delays skin cell changes caused by TPA.

Stress can cause hair loss by negatively affecting hair follicles and this effect might be reversed with specific treatments.

Targeting androgen receptors with antiandrogen oligonucleotides could effectively treat hair loss.

Overexpressing the mineralocorticoid receptor in mouse skin causes skin thinning, early skin barrier development, eye issues, and hair loss.

Estrogens can reduce DHT production in hair follicles, but are less effective than finasteride and progesterone.

Differences in androgen receptor expression and tissue properties may lead to higher cryptorchidism risk in certain rats.

Hairless protein is key for hair growth, cell differences cause gene expression variation, and the N-end rule pathway senses nitric oxide for protein breakdown.

Nexin 1 helps control hair growth in young rats.

Edar signaling is crucial for proper hair follicle development and function.

Retinoic acid causes gland formation instead of hair in mouse skin by altering epidermal and dermal interactions.

A woman with a new PTCH gene mutation has both Gorlin syndrome and severe hair loss.

Stem cells can create hair follicles, potentially treating permanent hair loss, and healthy skin and hair depend on mitochondrial function and special fats.

Patients with certain FoxN1 gene mutations have severe immune issues but normal skin and hair.