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Patients with certain FoxN1 gene mutations have severe immune issues but normal skin and hair.

Nestin-positive cells are important for hair follicle regeneration in alopecia areata.

PADI4 enzyme slows down cell growth in developing hair follicles.

17α-estradiol boosts aromatase activity in female hair follicles, potentially helping with hair loss.

Scientists found new nonsteroidal compounds that can act like natural male hormones and might help treat male fertility and hormone issues.

Testosterone and some of its forms can strongly stimulate oil gland growth in skin.

EGFR is essential for normal hair development and follicle differentiation.

New compounds were made that block an enzyme linked to breast cancer better than existing treatments.

Activin A increases inner ear hair cell development, while follistatin decreases it.

Progesterone and its metabolites can prevent seizures without major side effects.

Keratin gene regulation is similar across mammals, affecting hair follicle differentiation.

Rat whisker cells can help turn other cells into nerve cells and might be used to treat brain injuries or diseases.

New treatments for cancer and skin disorders show promise in disrupting harmful cell interactions and promoting hair growth.

These drugs cause hair growth without affecting testosterone or adrenal hormones.

Cortisol increases a specific protein in bone cells, which may help explain its negative effects on bone.

Estrogen increases blood vessel growth factor production, while testosterone blocks this increase.

Stress may affect hair growth by influencing hair follicle development and could contribute to hair loss.

Androgens are essential for maintaining adult neuromuscular structure.

17β-estradiol may help hair growth by increasing cannabinoid receptor type 1.

Prenatal retinoic acid exposure increased cell proliferation in mouse hair follicles without affecting their development.

The same genetic mutation in Sjögren-Larsson syndrome can lead to different levels of severity, suggesting other factors influence the symptoms.

Blue-light activation of TrkA improves hair-follicle stem cells' ability to become neurons and glial cells.

Certain genetic variants reduce enzyme activity, contributing to non-classic congenital adrenal hyperplasia.

ODC overexpression in hair cells increases tumor growth by reducing Notch signaling.

The ACTH/MC2R system is crucial for controlling hair growth cycles in mice.

Antisense oligonucleotides can reduce androgen receptor levels in skin cells, suggesting potential for treating skin disorders.

Keratin expression reflects cell organization and differentiation, not causes it.