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ERK activation spreads between cells in mouse skin, linked to cell division and influenced by TPA and EGF receptors.

Nfib in hair follicle stem cells boosts melanocyte stem cell growth and differentiation.

A new protein was made to detect specific skin cell growth receptors and worked in normal skin but not in skin cancer cells.

TGF-β and FGF pathways are crucial for skin development and regeneration.

Dkk4 is necessary for the initial development and arrangement of hair follicles.

FLCN helps control iron levels in cells.

Disrupting the Flcn gene in mice causes early kidney cysts and tumors, which can be treated with rapamycin.

KGF and its receptor are found in enlarged prostate tissue and KGF strongly increases cell growth.

Activating PKCα in skin causes cell death and inflammation through different pathways.

Folliculin deficiency causes problems with cell division and positioning due to disrupted RhoA signaling and interaction with p0071.

Targeting non-Smad pathways in TGF-β signaling may improve keloid treatment.

NFIC helps rat dental cells grow and turn into bone-like cells.

Dkk4 protein is necessary for the proper development and arrangement of hair follicles.

Igf1r helps regulate hair growth cycles.

Nod factor can trigger changes in legume root hairs with just one molecule.

BFNB could be a promising treatment for hair growth.

Overexpression of PKCepsilon leads to increased TNFalpha, promoting metastatic squamous cell carcinoma in mice.

Deleting the CD271 gene in mouse skin cells leads to disorganized skin and increased hair growth, suggesting CD271 is important for skin health.

Fibronectin is essential for hair follicle regeneration by supporting stem cells.

Certain mutations in the KLHL24 gene cause a skin disorder by breaking down an important skin protein.

High levels of ERK activity are key for tissue regeneration in spiny mice, and activating ERK can potentially redirect scar-forming healing towards regenerative healing in mammals.

Mathematical modeling helps understand and predict the MAPK cell signaling pathway.

TGF-β signaling is essential for new hair growth after wounds.

Ku80 is a key receptor for Thymosin β4, affecting cell migration and wound healing.

The LEF-1 binding site enhances gene expression in hair follicles, with other proteins aiding specific regulation.

TGF-β signaling is essential for hair follicle regeneration during wound healing.

Noggin promotes skin tumors by activating Wnt and Shh pathways.

PNKP is essential for keeping adult mouse progenitor cells healthy and growing normally.

Keratinocyte growth factor significantly alters skin and tissue development.