Search

everythinglearnresearchcommunity

for

Search:↓

Immune cells in Hidradenitis suppurativa become more inflammatory and may be important for treatment targets.

Resident memory T cells and necroptosis may drive fibrosis in eosinophilic fasciitis and morphea.

RSPO1 could help create new diabetes treatments by increasing pancreatic β cells.

UC.145 may be a new biomarker for predicting gastric cancer.

ASLAN004 was safe and well-tolerated, supporting further development for treating certain diseases.

Extracellular nucleic acids help start inflammation needed for tissue repair, but must be properly regulated.

Not having the gene PLAAT3 leads to fat loss, high insulin resistance, and abnormal fat levels in the blood due to a disruption in fat cell development and function.

Rapamycin treatment helps reduce brain inflammation and symptoms of mitochondrial disease by blocking specific pathways in mice.

SPT6 prevents excessive skin inflammation by blocking a feedback loop.

Certain genetic markers can indicate higher or lower risk for systemic lupus erythematosus.

IL-24 contributes to skin aging by increasing collagen-degrading enzymes and decreasing collagen production when exposed to air pollution.

Four specific genes are linked to keloid formation and could be potential treatment targets.

Acne patients' skin, both with and without lesions, shows a strong immune response and higher antimicrobial activity.

RpoS helps Borrelia burgdorferi survive in hosts and adapt to different environments.

Par3–mInsc and Gαi3 work together to ensure proper cell division orientation in skin development.

RANKL improves the immune response against herpes simplex virus by enhancing T cell activation and could help develop better treatments or vaccines.

Scarred skin in lichen planopilaris loses immune cells due to a decrease in a specific protein in skin cells.

Mrp3 helps in wound healing and hair growth.

FLRG and follistatin have different roles in wound healing.

Type 2 inflammation helps wound healing by switching immune cells to repair mode.

ERULUS controls root hair growth by regulating cell wall composition and pectin activity.

P2X₃-IR fibers are widespread in rat skin and likely help detect pain.

CTHRC1 is essential for healing and preventing heart rupture after a heart attack.

Targeting TRP channels may help reduce excessive scarring.

NEDD4 and NEDD4L help control intestinal stem cells and prevent tumors by breaking down the LGR5 receptor.

Activating TLR9 helps heal wounds and regrow hair by using specific immune cells.

Scalp psoriasis inflammation is mainly caused by bacteria-related triggers.

Lygodium japonicum spores can reduce inflammation by blocking certain cell signals.