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Researchers fixed gene mutations causing a skin disease in stem cells, which then improved skin grafts in mice.

RXRa is crucial for Th2 immune cell development and may link nutrition to immune health.

Patients with the same vitamin D receptor mutation showed different symptoms due to other factors.

CAP1/Prss8 does not activate PAR2 or inhibit PN-1.

Protease Nexin-1 is found in human hair growth cells and is affected by male hormones.

Researchers found key regions in the mouse hairless gene that control its activity in skin and brain cells, affecting hair follicle function.

The study concluded that specific proteins are necessary to maintain the structure that holds epithelial cells tightly together.

RNase L hinders hair growth by altering immune signals.

Mutations in the NR3C1 gene cause a rare condition that affects hormone signaling and can lead to various symptoms, with dexamethasone as a treatment option.

Noncoding dsRNA boosts hair growth by activating TLR3 and increasing retinoic acid.

FLCN helps control iron levels in cells.

Increasing Rps14 helps grow more inner ear cells and repair hearing cells in baby mice.

Vitamin D receptor can control the hairless gene linked to hair loss even without vitamin D.

Nuclear Factor I-C is important for controlling hair growth by affecting the TGF-β1 pathway.

A specific protein complex increases the activity of a plant enzyme, but this action is not required for plant root hair growth.

The near-naked hairless mutation causes hair loss but is not due to a mutation in the hairless gene itself.

A KLK5 inhibitor effectively improved skin symptoms in a mouse model of Netherton Syndrome.

A specific RNA, circNlgn, contributes to heart damage and scarring caused by the cancer drug doxorubicin.

The mutant HR bmh protein affects hair follicle formation by failing to repress vitamin D receptor activity.

A new genetic disorder caused by an ODC1 mutation can be treated with DFMO.

RXR and RAR proteins in skin may help with cell growth, hair growth, and gland function.

FGF9 controls which receptors it binds to through a process where two FGF9 molecules join, and changes in FGF9 can lead to incorrect receptor activation.

KRT2.13 gene is similar to KRT2.9 but not expressed in hair follicles.

Measuring 24-OHase induction helps identify defects in vitamin D processing and predict treatment response.

A vitamin D receptor mutation causes rickets and affects immune responses.

Mice lacking steroid 5α-reductase 2 show less aggression and better impulse control.

GTL1 is needed to control root hair growth and prevent problems when there are too many nutrients.

Normal skin cell renewal doesn't need RAR signaling, but vitamin A-related skin thickening does.

Mutations in the FOXN1 gene cause severe immune issues but don't affect hair and nails.