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Protein kinase C epsilon may promote skin cancer development after UV exposure by affecting nearby cells.

Poly(ADP-ribose) synthesis is linked to skin cell differentiation.

TET enzymes are important for skin and hair development by controlling gene activity in specific areas.

Genotyping for NUDT15 p.Arg139Cys can help predict thiopurine side effects in Japanese IBD patients.

TAp63 and NRF2 work together to manage oxidative stress, preventing premature aging and aiding skin functions.

RNase L hinders hair follicle regeneration by altering immune signals.

Increasing SSAT makes skin more prone to cancer.

Noncoding dsRNA helps produce exosomes that aid in skin regeneration.

Toll-like receptor 3 helps repair the skin barrier after UV damage.

Keratinocytes can reduce the survival of certain melanoma cells, suggesting new therapy paths.

The DNMT3B -579G>T polymorphism may increase the risk of colorectal cancer.

The reconstructed skin model from hair follicles functions like human skin in processing chemicals and can be used to test ingredient safety.

A new mutation in the XEDAR gene might cause a rare skin condition called hypohidrotic ectodermal dysplasia.

Mobile phone radiation can cause DNA damage in human hair root cells.

Trichorrhexis nodosa is mainly caused by hair trauma, not a metabolic defect.

α-type filaments in guinea pig hair follicles have unique structural features.

Podiatrists perform nearly all nail excisions in the US.

Using special RNA to target a mutant gene fixed hair problems in mice.

miR-214-3p helps nerve repair and recovery.

The microenvironment affects the behavior and survival of melanocytes with the GNAQ oncogene in melanoma.

A rare gene variant causes hair and nail issues in a family.

A mutation in the KRTHB5 gene causes hair and nail issues.

A variant in the KRT31 gene causes a rare hereditary hair disorder called monilethrix.

Vitamin D may help protect against nonmelanoma skin cancer.

The type of tumor suppressor gene lost affects the behavior of skin cancer.

Deleting the MAD2L1 gene in mice led to rapid tumor growth despite chromosomal instability.