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Removing a methyl group from the ITGAV gene speeds up bone formation in a specific type of bone disease model.

Ablative fractional laser treatment nearly matches the gene reduction effects of topical vismodegib in skin cancer.

K6 gene expression can be controlled and manipulated in mice for studying skin disorders.

Nano-PROTACs could improve drug targeting and delivery by using nanotechnology.

The K5 promoter controls gene expression in skin cells, with specific DNA segments crucial for targeting and regulation.

Screening for the NUDT15 gene variant can prevent severe side effects from thiopurine drugs in East-Asian people.

Researchers fixed gene mutations causing a skin disease in stem cells, which then improved skin grafts in mice.

A new genetic mutation in the ODC1 gene causes developmental delay and other symptoms in a young girl.

Targeted photodynamic therapy is a promising method for precise disease treatment and diagnosis.

Introducing the rat OTC gene normalized hair growth in SPF-ASH mice.

The study mapped keratin 15 and 19 genes, aiding future genetic disorder research.

Engineered cytokines show promise for improving tissue healing and safety in regenerative medicine.

Overexpressing SSAT in mice makes them highly sensitive to polyamine analogues, causing liver damage and high mortality.

Researchers found a non-functional sheep keratin gene due to mutations.

TET enzymes are important for skin and hair development by controlling gene activity in specific areas.

Suppressing ODC activity reduces tumor growth in hair follicles.

High levels of ODC and a mutant Ha-ras gene cause tumors in mice.

Deleting the MAD2L1 gene in mice led to rapid tumor growth despite chromosomal instability.

Researchers found a safe and effective way to pick genetically modified skin cells with high growth potential using CD24.

Tet2 and Tet3 enzymes are important for controlling hair growth and shape by affecting gene activity and DNA structure in hair follicles.

TPA strongly increases ODC activity in certain skin cells, potentially aiding tumor growth.

ANE syndrome is caused by a mutation in the RBM28 protein that disrupts ribosome assembly.

Knocking out certain genes in mice helps understand skin and hair growth problems.

Sheep cells were successfully modified to include a spider silk protein gene.

The tumor suppressor gene FLCN affects mitochondrial function and energy use in cells.

Researchers found that most genes affecting drug responses are not fully covered by commercial SNP chips, suggesting the need for more comprehensive tools to optimize drug selection based on genetics.

The plant hormone auxin activates the TOR pathway, affecting gene expression related to growth and cell size.

MT-DADMe-ImmA can selectively kill head and neck cancer cells without harming normal cells.