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RSPO1 mutations in certain patients lead to skin cells that don't develop properly and are more likely to become invasive, increasing the risk of skin cancer.

A specific gene mutation causes woolly hair and hair loss.

Keratin 17 gene mutations cause both steatocystoma multiplex and pachyonychia congenita type 2.

NCSTN gene mutation causes abnormal skin cell differentiation and more inflammation, contributing to Hidradenitis Suppurativa.

Deleting TNFα gene reduces skin cancer risk in certain mice.

A gene mutation causes curly hair and hair loss in rats.

Inhibiting AP-1 changes skin tumor types and affects tumor cell identity.

Mice with extra PKCδ resist chemical-induced skin cancer but not UV-induced.

HPV8 causes skin cancer by expanding specific skin stem cells.

Inhibiting AP-1 changes skin tumor types and affects tumor cell identity.

Alpha-difluoromethylornithine prevents cancer in mice but causes hair loss.

SOX4 is crucial for the development of melanoma.

A PIK3CA mutation in Schwann cells causes severe nerve damage and increased glycolysis, but early treatment can help.

Increased ODC expression makes normally tumor-resistant mice more prone to tumor development.

The HCR gene contributes to psoriasis risk.

Mutations in specific keratin genes cause improper hair structure in mice due to faulty keratin protein assembly.

A new genetic mutation linked to CARASIL syndrome and small artery disease was found in a Chinese family.

Deleting vitamin D and calcium receptors in skin cells increases skin cancer risk by reducing DNA repair and stress response.

Krox20 is crucial for hair growth and maintaining skin stem cells.

Loss of Pten in certain hair follicle stem cells increases skin cancer risk.

Protein Kinase Cε increases skin sensitivity to UV damage and skin cancer risk.

Overexpressing COX-2 in mice skin reduces skin tumor development.

A mutation in the KRT25 gene causes woolly hair and hair loss.

Keratin 17 is modified by RSK1 in response to growth and stress, affecting skin growth and stress response.

ETS2 is crucial in squamous cell carcinoma development and could be a therapeutic target.

A new gene location for Keratosis follicularis squamosa was found on chromosome 7p14.3-7p12.1.

A new mutation linked to partial Androgen Insensitivity Syndrome and prostate cancer was found in a patient unhappy with their female gender assignment.

PRSS35 enzyme may help start skin tumors and could be a target for cancer treatment.

ODC overexpression in hair cells increases tumor growth by reducing Notch signaling.

Breast cancer treatments work better with AR activation, improving results and reducing side effects.