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A new gene mutation causes vitamin D resistance and hair loss in two unrelated girls.

PDGF signaling may play a role in hair growth cycle regulation.

PRP can improve hair growth in people with hair loss, but more research is needed.

Prostaglandin D2 blocks new hair growth after skin injury through the Gpr44 receptor.

VEGF stimulates hair cell growth and increases growth receptor levels through a specific signaling pathway.

Measuring 24-OHase induction helps identify defects in vitamin D processing and predict treatment response.

PGD2 increases androgen receptor activity in hair cells, which could be targeted to treat hair loss.

ACTH may be an effective first-line treatment for acute calcium pyrophosphate crystal arthritis.

Mice without active or present vitamin D receptors maintain normal blood sugar control and islet gene expression when calcium levels are normal.

Vitamin D receptor helps control hair growth and could be used to treat certain skin tumors.

A mutation in the vitamin D receptor causes severe resistance to vitamin D, affecting bone health but not hair growth.

A woman with a rare ovarian tumor and hyperparathyroidism improved after surgery, highlighting the need for reporting unusual cases to understand and manage rare diseases.

Sex steroids influence skin appendage function and neoplasm development, with changes in AR and ERalpha in certain gland neoplasms.

Calretinin identifies the companion cell layer in human hair follicles.

Overexpression of PKCepsilon leads to increased TNFalpha, promoting metastatic squamous cell carcinoma in mice.

TRPV4 helps cells repair tissue and reduce scarring by controlling calcium levels.

Nuclear hormone receptors play a significant role in skin wound healing and could lead to better treatment methods.

ROR2 is essential for hair follicle stem cell renewal and maintenance.

Activating ER-β, not ER-α, improves skin cell growth and wound healing.

TRPV3 channels are involved in skin processes and are affected by shear stress, influencing itch and mechanotransduction.

Lack of vitamin D receptor increases skin tumor risk by boosting hedgehog signaling.

Cortisol increases a specific protein in bone cells, which may help explain its negative effects on bone.

Rathke’s cleft cysts can rarely cause Cushing disease, and surgery can improve symptoms.

TLR2 is crucial for hair growth and regeneration, and boosting it can help prevent hair loss.

The antibody 005-C04 blocks prolactin receptors, causing reversible infertility, impaired lactation, and hair regrowth in female mice.

Vitamin D receptor actions without binding are crucial for healthy skin and hair.