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Zfp157 is active in many mouse tissues during development and in specific adult cells.

A specific gene change in APCDD1 increases the risk of hair loss.

α-Phellandrene may help prevent hair loss by increasing growth factors and cell growth in hair cells through a specific signaling pathway.

Retinoic acid, glucocorticoids, and IGF1 increase IGFBP-3 production in human dermal papilla cells, affecting hair growth.

Inward rectifier potassium channels are important in many body functions and diseases, and could be potential drug targets.

MPZL3 protein is important for controlling hair growth cycles.

PP405 may help hair growth by activating hair follicle stem cells.

New compounds may help treat heart disease by activating specific potassium channels.

The flaky skin mouse mutation is a natural model for studying human psoriasis.

CaBP1 and CaBP2 are important for maintaining hearing by supporting continuous calcium currents and nerve signaling in the ear.

Keratin films from human hair can potentially replace human nail plates for drug testing.

Hyaluronate fragments can help reverse skin thinning by working with the CD44 receptor.

Key signals for hair follicle formation were identified.

The UHS promoter is specific to mouse hair follicles.

CCC1 is essential for ion balance and proper plant cell function.

Mice with too much Claudin-6 have skin barrier problems and abnormal hair growth.

A certain gene variation can affect protein production and is linked to male pattern baldness.

ATP-dependent chromatin-remodeling complexes are crucial for gene regulation, cell differentiation, and organ development in mammals.

Inflammation in psoriasis may trigger antimicrobial peptides and cell death.

Certain peptides can prevent hair loss in young rats caused by a cancer drug.

Keratin proteins are crucial for hair growth and structure.

Water extract of Cacumen Platycladi helps hair growth by activating specific cell pathways.

Pig skin cells can turn into mesodermal cells but lose their ability to become neural cells.

Overexpression of Hoxc13 in hair cells causes hair loss and skin issues.

SOX11 and SOX4 help skin cells act like embryonic cells to heal wounds in mice.

Noggin overexpression delays eyelid opening by affecting cell death and skin cell development.

HSD11b1 affects skin nerves and increases non-histaminergic itch.

CRISPR/Cas9 and prime editing can potentially fix skin disorder genes safely and effectively.

Normal human melanocytes can avoid cell death through multiple pathways.