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The Hippo signaling pathway helps control organ size during regeneration by regulating gene expression.

Basonuclin may help control ribosomal RNA gene activity in skin cells.

Increasing Rps14 helps grow more inner ear cells and repair hearing cells in baby mice.

MCPIP1 in myeloid cells is important for skin cancer development and healthy hair growth.

Understanding gene expression in hair follicles can reveal insights into hair growth and disorders.

hsa_circ_0002980 can help stop liver cancer cells from growing and spreading.

Dual TCR Treg cells are common in mouse tissues and vary by location.

A specific gene segment can make mouse skin cells glow, helping study hair growth and gene effects.

The white wax scale insect's genome shows that complete metamorphosis evolved earlier than thought and highlights differences in male and female development.

Trps1 is essential for proper hair follicle development.

PKM2 is a promising target for heart repair and regeneration.

Proper niche formation in Drosophila requires Slit-Robo signaling for cell migration.

Ectodysplasin-A1 is crucial for developing hair, teeth, and glands.

TCDD changes skin cell growth and keratin production in mice.

Vitamin D receptor is essential for hair growth signaling.

Epiprofin helps cells grow in developing teeth, hair, and limbs.

Inhibiting ornithine decarboxylase may help prevent certain skin cancers.

Early development of hair, teeth, and glands involves specific signaling pathways and cellular interactions.

Polyomavirus A2 infection in newborn mice caused hair follicle tumors.

FOXN1 duplication can cause excessive hair growth.

Certain interactions help prepare the androgen receptor for pairing and activation, which is important for its role in development and disease.

RORA plays a key role in controlling seasonal hair molting by affecting hair follicle cell activity.

Multiple mouse desmoglein 1 isoforms have distinct roles in skin and hair development.

H19 may help cause PCOS by affecting CTGF levels, suggesting a new treatment target.

Removing PLCg1 from skin cells caused thicker oil glands and less hair in mice.

Loss of Dnmt3a and Dnmt3b leads to more aggressive skin tumors, but blocking PPAR-γ can reduce this effect.

Overexpression of certain genes can shorten hair by disrupting the hair-growth cycle.

Increased ODC activity leads to skin tumors by recruiting stem cells, not by toxic byproducts.

Some men's prostate tissues have low enzyme levels due to genetic changes, possibly affecting treatment for prostate enlargement.