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Blocking a key energy pathway in human hair follicles can trigger stress responses that stop cell growth.

FGF9 controls which receptors it binds to through a process where two FGF9 molecules join, and changes in FGF9 can lead to incorrect receptor activation.

Patients with the same genetic mutation for vitamin D-resistant rickets showed different symptoms but all improved with treatment except for hair loss.

PRC1 influences skin stem cell development by both turning genes on and off, affecting hair growth and skin cell types.

α-parvin is necessary for skin and hair growth and for the correct orientation of skin cells.

CAP1 helps Arabidopsis plants grow better under ammonium stress.

Retinoic acid-binding proteins in skin are regulated by β-catenin and Notch signalling.

RXRα is crucial for proper immune response and links diet to immune function.

Certain peptides can prevent hair loss in young rats caused by a cancer drug.

Formyl-peptide receptor agonists could be new anti-inflammatory drugs.

A unique gene mutation causes vitamin D-resistant rickets without causing hair loss.

A genetic mutation in the LIPH gene causes a rare hair disorder with sparse, curly hair.

PKM2 is a promising target for heart repair and regeneration.

A new mutation in the Hr gene causes hair loss in mice, similar to a human hair disorder.

Wnt10b promotes hair growth, while SFRP2 inhibits it in Wanxi Angora rabbits.

The KRTAP22-2 gene in sheep does not significantly affect wool traits.

TRPS1 loss in balding scalp areas contributes to hair loss in androgenic alopecia.

A Chinese family had a child with a specific gene mutation causing vitamin D-resistant rickets, but the child improved with calcium and low-dose calcitriol.

The study concludes that the EDA2R gene is activated by p53 during chemotherapy but is not necessary for chemotherapy-induced hair loss.

A new treatment using a DNA aptamer can promote hair growth by targeting a specific receptor.

Localized ROS production is essential for cell growth and movement in plants and animals.

Adults with Tatton-Brown-Rahman syndrome may have serious heart problems and need lifelong heart monitoring.

A gene called APCDD1, which controls hair growth, is found to be faulty in a type of hair loss called hereditary hypotrichosis simplex.

FERONIA regulates plant growth, pollen interactions, and sugar signaling.

Mutations in the AP1B1 gene cause a new syndrome with skin, hearing, and developmental issues.

High energy boosts root hair growth in plants, while low energy stops it.

Local positive feedback helps shape root hair cells by stabilizing growth sites.

New mutations in the hairless gene may cause hair loss and affect bone development.

The BMP2 gene is more active in the early growth phase of Cashmere goat hair and may affect hair regeneration and textile production.

Mutations in the KRT16 gene can cause skin and nail disorders.