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Cis-UCA can form new compounds without enzymes, and sulphide donors reduce its UVB-induced toxicity in skin cells.

Ca²⁺-mediated protein citrullination controls cell growth in the CNS and may help treat brain tumors.

Compounds from Pterocarpus indicus may help treat benign prostatic hyperplasia by stopping cell growth.

Collagen XVIII affects wound healing, hair growth, and bone development, with its absence speeding up processes and overexpression causing delays and abnormalities.

Deleting vitamin D and calcium receptors in skin cells increases skin cancer risk by reducing DNA repair and stress response.

Coinheritance of BRCA2 and CYLD genes may lead to new treatment options for certain cancers.

Apoptotic cells may trigger cell death in hair follicles during their regression cycle.

CENPV, a new partner of CYLD, helps regulate ciliary acetylated tubulin and is overexpressed in certain skin tumors.

Resident memory T cells and necroptosis may drive fibrosis in eosinophilic fasciitis and morphea.

A patient with a PLEC mutation has epidermolysis bullosa, muscular dystrophy, and myasthenia gravis, which improved with steroid treatment.

The study concluded that a protein important for hair strength is regulated by certain molecular processes and is affected by growth phases.

Pilomatrixoma involves abnormal hair keratin production and cell death, causing debris and cysts.

Researchers found a specific immune receptor in patients that causes severe skin reactions to a drug.

The study created a mouse model to mimic degenerative diseases for testing tissue repair and new therapies.

Overexpressing PKCepsilon in mice reduces papillomas but increases carcinomas.

Decreasing MIG6 can increase the movement and invasiveness of MEK-inhibited mutant NRAS melanoma, particularly when stimulated by EGF.

CRISPR/Cas9 has improved precision and control but still faces clinical challenges.

Different melanocyte types in hair follicles either survive or die during the catagen phase.

Certain CD8+ T cells attack hair follicles in alopecia areata, suggesting they could be targeted for treatment.

CD8+ T cells are involved in alopecia areata and may cause disease relapse.

A new therapy for a skin blistering condition has not been developed yet.

CRISPR gene editing reduces harmful molecules in cells from Emery–Dreifuss Muscular Dystrophy patients.

Low-dose 800 nm laser effectively treats blackheads and coarse pores without harming cell growth.

AP collagen peptides may help hair grow better and become stronger.

Mutations in the desmoplakin gene can cause hair problems and deadly heart disease.

The study found that analyzing certain cell signaling pathways is not a reliable method to tell apart two types of skin tumors.

A gene variant causes patched hair loss in mice, similar to alopecia areata in humans.

Double-stranded RNA activates a pathway that causes a skin protein to be expressed in the wrong place.