Search

everythinglearnresearchcommunity

for

Search:↓

JAK/STAT1 pathway causes hair loss during chemotherapy by reducing Shh in hair follicles.

Lack of Ctip2 in skin cells delays wound healing and disrupts hair follicle stem cell markers in mice.

Protein kinase C epsilon may increase skin cancer risk by affecting nearby cells.

Blocking a protein called CXXC5 with a specific peptide can stimulate hair regrowth and new hair growth in wounds.

Researchers created a mouse with the same mutation as humans with trichothiodystrophy, showing similar symptoms and confirming the condition is due to defects in DNA repair and gene activity.

Removing Ctip2 in skin cells causes skin inflammation similar to atopic dermatitis.

CRABP2 helps increase the growth of cells important for hair growth by activating a specific growth pathway.

Deleting TNFα gene reduces skin cancer risk in certain mice.

Mutant CDP/Cux protein causes hair defects and reduced male fertility in mice.

WWP2 is crucial for tooth development in mice.

SOX18 helps sheep hair cells grow by activating a specific cell growth pathway.

Activating the PI3K/Akt pathway improves hair growth by human dermal papilla cells in hair beads.

TSC2 is crucial for proper hair follicle development and patterning.

NCSTN gene mutation causes abnormal skin cell differentiation and more inflammation, contributing to Hidradenitis Suppurativa.

Inactivating β-catenin is essential for chick retina regeneration.

Overexpressing CtBP1 in skin cells causes skin and hair problems.

Hedgehog signaling is crucial for hair development, cadherins affect cell adhesion, neutrophils play a role in skin lesions, and BP230 autoantibodies impact skin stability.

P-cadherin is important for hair growth and health, and its problems can cause hair and skin disorders.

Activating β-catenin can turn skin cells into hair follicles.

Beta-catenin boosts hair follicle cell growth by increasing c-myc expression.

Sonic hedgehog signaling is crucial for normal wound healing.

EGFR helps mouse hair follicles stop growing by reducing certain growth regulators.

Wnt and Shh signaling are key in noggin-induced tumors, and blocking them can slow tumor growth.

Activating Wnt signaling improves the efficiency and safety of creating stem cells.

Ptprq has multiple forms that change during inner ear development.

Deleting the PTEN gene in mice causes nerve cells to grow larger and heal better after injury, but may cause overgrowth and hair loss in older mice.

CTCF protein is essential for skin and hair follicle development in mice.

Polycomb Repressive Complex 2 is not needed for hair regeneration.

The peach gene pCTG134 helps control the interaction between auxin and ethylene hormones during fruit ripening.

Faulty Notch signalling may cause hair follicle changes and inflammation in hidradenitis suppurativa.