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Removing β-catenin in certain stem cells causes hair whitening and pigmentation issues.

Overexpression of hurpin in mice leads to abnormal skin and higher skin cancer risk.

Increased ODC expression makes normally tumor-resistant mice more prone to tumor development.

A PKP1 gene mutation causes skin fragility and hair loss in Chesapeake Bay retriever puppies.

Introducing the rat OTC gene normalized hair growth in SPF-ASH mice.

A mouse gene mutation increases the risk of skin cancer.

Introducing the OTC gene improved symptoms in mice with OTC deficiency.

Deleting TNFα gene reduces skin cancer risk in certain mice.

PKC ε increases hair follicle stem cell turnover and may raise skin cancer risk.

The HtrA1L364P mutation causes brain dysfunction and blood vessel damage.

A chimeric keratin partially improved skin structure in mice lacking keratin 5, but didn't fully restore normal skin.

Overexpression of PTHrP in chondrocytes causes short-limbed dwarfism and delayed bone formation in mice.

Tabby mutations in mice affect hair follicle development and help study genetic mapping and certain medical conditions.

Activated β-catenin affects hair growth and skin thickness, and changes are reversible.

Deleting Smad4 and PTEN genes in mice causes rapid, invasive forestomach cancer.

Stress can cause a type of hair loss in mice lacking the CCHCR1 gene.

Zfp462 deficiency in mice causes anxiety-like behaviors and excessive self-grooming.

Inactivating both p53 and Rb genes in mice speeds up aggressive skin cancer development.

Blocking Wnt signaling in young mice causes thymus shrinkage and cell loss, but recovery is possible when the block is removed.

Deleting the MAD2L1 gene in mice led to rapid tumor growth despite chromosomal instability.

Cupric chloride treatment corrected abnormal Purkinje cell development in brindled mice.

The mutation causing Hutchinson-Gilford progeria syndrome leads to severe skin problems and early death in mice.

Deleting Smad4 and PTEN genes in mice causes rapid, invasive stomach cancer.

Lack of Ctip2 in skin cells delays wound healing and disrupts hair follicle stem cell markers in mice.

Msx2 deficiency in mice leads to bone growth and organ development problems.

FTase and GGTase-I are essential for skin keratinocyte health.

Mutant MK6a transgenes in mice cause blistering, hair loss, and potential human alopecia.

A gene mutation in mice causes hair loss, weak bones, and protein buildup, showing how protein processing issues can lead to diseases.

A mutation in mice causes hair loss and skin issues due to a defect in a gene affecting cell adhesion.