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Overexpression of SSAT causes hair loss and skin issues, but reducing putrescine can help.

PAK4 is crucial in cancer progression, brain development, and could be a therapeutic target, especially through the PAK4-CREB axis.

CAP1/Prss8 does not activate PAR2 or inhibit PN-1.

TPA strongly increases ODC activity in certain skin cells, potentially aiding tumor growth.

Activating PKM2 and Wnt/β-catenin signaling together can potentially enhance hair growth and could be a treatment for hair loss.

Grasp protein helps maintain skin health after UVB exposure.

Pyruvate Kinase M2 helps hair grow by linking energy production and a key hair growth pathway.

Scientists discovered a new way UVB light increases skin pigmentation through the ATP-P2X7 pathway.

Spt4 and Spt6 are essential for fat cell development.

The mTOR pathway may be involved in the development of hair follicle tumors, with higher activity in malignant tumors.

Protein kinase C epsilon may promote skin cancer development after UV exposure by affecting nearby cells.

Blocking Oncostatin M's role in the JAK-STAT pathway can stimulate hair growth in mice.

The enzyme pyruvate kinase M2 helps hair regrowth and could be a potential treatment for hair loss.

Astrotactin2 affects hair follicle orientation and skin cell polarity.

Overexpression of SSAT in mice causes hair loss, liver damage, and sensitivity to polyamine analogues.

Beta-catenin boosts hair follicle cell growth by increasing c-myc expression.

p2y5, now called LPA6, is a receptor important for human hair growth.

PSAT1 is key for hair growth and stem cell function in cashmere goats.

PNKP is essential for keeping adult mouse progenitor cells healthy and growing normally.

Astrotactin-2 is cleaved in a specific way that helps understand its maturation.

mrp/plf-mRNA can indicate tumor-promoting effects in skin.

Blocking mTORC1 activity could increase hair pigmentation and potentially reverse greying.

The plant hormone auxin activates the TOR pathway, affecting gene expression related to growth and cell size.

Rapamycin treatment helps with mitochondrial disease by reducing PKC levels.

Rapamycin may help treat Leigh syndrome by targeting protein kinase C.

Protein kinase C epsilon may increase skin cancer risk by affecting nearby cells.

PHGDH promotes cancer growth by aiding cell proliferation and tumor progression.

SPT6 prevents excessive skin inflammation by blocking a feedback loop.

Plasmalogens activate a channel in cells that may stimulate hair growth.