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Prostaglandin D2 increases testosterone production in skin cells through a process involving reactive oxygen species, which could be a new target for treating hair loss and other skin conditions driven by testosterone.

A new mutation in the TRPS1 gene caused Trichorhinophalangeal syndrome in a 17-year-old, highlighting the need for genetic testing.

Scientists identified three genes important for processing certain brain chemicals, thyroid hormones, and medications.

The system allows precise control of gene expression in mouse skin, useful for studying skin biology.

Lipocalin-Type Prostaglandin D2 Synthase (L-PGDS) is a protein that plays many roles in the body, including sleep regulation, pain management, food intake, and protection against harmful substances. It also affects fat metabolism, glucose intolerance, cell maturation, and is involved in various diseases like diabetes, cancer, and arthritis. It can influence sex organ development and embryonic cell differentiation, and its levels can be used as a diagnostic marker for certain conditions.

TSPO might help treat anxiety and depression.

SFRP2 and PTGDS may be key factors in female hair loss.

A woman with a unique syndrome similar to TRPS has a genetic change near the TRPS1 gene, affecting its regulation.

Activated ras can protect kidney cells from a certain substance that causes cell death.

Mice studies show that Protein Phosphatase 2A is crucial for cell growth, development, and disease prevention.

Five different TPO gene mutations were found in PCOS patients with thyroid hormone issues.

Mouse high-glycine/tyrosine proteins have distinct patterns in hair follicles, peaking at specific hair cycle days.

The study found a specific peptide that helps detect TGase 3 activity in skin and hair follicles.

Increasing TSPO in the brain reduces anxiety and depression.

TRPS1 is crucial for bone, kidney, and hair follicle development.

Human and mouse TGase3 enzymes are similar but differ near the activation site, crucial for their function in skin and hair development.

Disrupting the Tsc2 gene in certain cells leads to thicker skin, larger hair, and changes in hair growth signaling, which can be partly reversed with specific treatment.

Prostaglandin D2 increases testosterone production in skin cells through a process involving reactive oxygen species, and antioxidants may help treat hair loss.

TMPRSS2 helps viruses enter cells and protects the prostate from inflammation-related cancer.

Trps1 is essential for proper hair follicle development.

Personalized sonidegib dosing can effectively treat Gorlin-Goltz syndrome with fewer side effects.

Adding a small amount of TPGS to minoxidil can help hair growth, but too much TPGS reduces this effect and increases minoxidil in the blood.

Controlling transposable elements is crucial for successful tissue regeneration.

The Itpr3 gene causes a specific hair pattern in mice.

Blocking Prostaglandin D₂ (PGD₂) could help treat hair loss.

CE-PTG is a better method for analyzing hair growth in androgenetic alopecia.

The TGM3 gene's promoter region is key for skin and hair cell function and may aid gene therapy.

Researchers created a new mouse model, G4, that mimics human PCOS symptoms and links the condition to a specific gene.

Researchers created a mouse with the same mutation as humans with trichothiodystrophy, showing similar symptoms and confirming the condition is due to defects in DNA repair and gene activity.

The research suggests that SFRP2 and PTGDS proteins might be indicators of female pattern hair loss and could contribute to hair loss.