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Storing hair follicle micrografts for longer times can cause them to enter a state similar to the natural hair shedding phase, which might impact hair transplant results.

A new mutation in the K6b gene caused a girl's late-appearing nail condition.

Protein kinase C epsilon may increase skin cancer risk by affecting nearby cells.

Hair follicle stem cells change states with age, affecting hair growth and aging.

Epidermal cysts come from the skin, while pilar cysts come from hair follicles.

The tumor suppressor gene FLCN affects mitochondrial function and energy use in cells.

Mice with the naked gene have missing or abnormal hair cells.

Overexpressing CtBP1 in skin cells causes skin and hair problems.

Stat3 activation increases hair follicle progenitors but reduces bulge region stem cells.

FOXN1 mutations cause severe immunodeficiency, hair loss, nail issues, and thymus defects.

Autofluorescence in hair follicle stem cells can interfere with studies but may help isolate these cells.

Aging reduces stem cell activation, leading to hair loss in mice lacking a specific enzyme.

Notch signaling is crucial for specifying niche cells in Drosophila testis.

Certain tyrosine kinases may regulate hair growth and could help develop hair loss treatments.

Sebaceous gland atrophy and abnormal function may contribute to hair loss in psoriasis.

Porokeratotic eccrine and hair follicle nevus is a very rare skin disorder possibly caused by a GJB2 gene mutation.

Dermal EZH2 controls skin cell growth and differentiation in mice.

SKPs are similar to adult skin stem cells and could help in skin repair and hair growth.

Most women with polycystic ovary syndrome (PCOS) have menstrual issues, infertility, acne, and excessive body hair, with the most common type being the non-androgenic phenotype.

Sweat glands and hair follicles are determined by opposing signals, with BMPs promoting sweat glands and blocking BMPs leading to hair follicles.

Using bioreactors, scientists can grow more skin stem cells that keep their ability to regenerate skin and hair.

Alopecia areata patients show unique protein activity patterns, suggesting imbalanced signaling pathways.

Improper regulation of hair follicle processes causes hairlessness.

TGF-β and TNF influence hair follicle cell fate, with TNF being more effective in triggering cell death.

Sensory neuron and Merkel-cell changes in the skin happen independently during normal skin maintenance.

Nε-(carboxymethyl) lysine delays hair growth by blocking a key protein.

PDGFA/AKT signaling is important for the growth and maintenance of certain skin fat cells.

Not eating enough protein can cause hair roots to shrink and lose color, and hair to become thinner.

Keratin patterns in hair follicles help understand hair growth and potential hair and nail disorders.

Women with regular menstrual cycles and PCOS have linked kisspeptin and LH hormone patterns, unlike those with irregular cycles.