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Targeted siRNA therapy may be a promising treatment for KID syndrome by reducing mutant gene expression and improving cell communication.

Vitamin C deficiency changes gene expression, affecting skin and hair health.

mTORC2 is crucial for healthy skin barrier by regulating lipids and filaggrin.

Fgfr2b helps maintain healthy skin and prevent cancer.

MC-DNA vector-based gene therapy can temporarily treat CBS deficiency in mice.

Mice without the vitamin D receptor gene lose hair due to disrupted hair follicle cycles.

Hereditary factors cause hair loss in mice by affecting skin and hair follicle structure.

Naked-mouse hair lacks certain proteins and has less soluble fibril.

Id2 gene helps keep hair follicle stem cells inactive.

SKH1 hairless mice have identifiable epidermal stem cells with specific markers.

A new imaging method helps see and study touch nerve endings in mouse skin.

Beige and leaden pigment genes act within melanocytes, affecting pigment patterns.

Lgr5 and the vitamin D receptor are key in controlling skin inflammation and tumor risk in mice.

Genetically modified rats help reveal how vitamin D affects bone and skin health.

Overexpression of a specific receptor in mice skin causes skin thinning, early skin barrier formation, eye issues, and hair loss.

CXCR2 in skin cells promotes tumor growth.

Scientists created a cell model to study and find treatments for a skin disease called RDEB.

The tumor suppressor gene FLCN affects mitochondrial function and energy use in cells.

Dog skin with hair loss, when transplanted to mice, regrew hair, suggesting the hair loss cause is likely body-wide, not skin-specific.

TYK2 inhibition may help treat alopecia areata by promoting hair growth and reducing immune response.

Finasteride reduces certain behaviors caused by D1-like receptor agonists but not by D2-like receptor agonists in mice.

Decreasing MIG6 can increase the movement and invasiveness of MEK-inhibited mutant NRAS melanoma, particularly when stimulated by EGF.

Too much Sonic Hedgehog protein stops hair growth in embryos.

Injecting lentiviral vectors into early gestation mice effectively targets skin stem cells for potential gene therapy.

A new genetic mutation in the ODC1 gene causes developmental delay and other symptoms in a young girl.

Removing a specific gene in certain skin cells causes hair loss on the body by disrupting normal hair development.

Researchers used CRISPR/Cas9 to create a goat with a gene that increased cashmere production by 74.5% without affecting quality.

Hairless USP mice have enlarged skin cysts as they age.

Removing vitamin D and calcium receptors in mice skin cells slows down skin wound healing.