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Copper treatments increase copper in all tissues, but brindled female mice accumulate much more copper in their kidneys without clinical effects, unlike brindled male mice where brain copper deficiency is clinically significant.

The spiny mouse regenerates ear tissue asymmetrically, with gene expression differences possibly explaining its unique healing abilities.

African spiny mice can regenerate skin and hair after wounds due to specific tissue mechanics.

Twist2 is essential for proper skin healing and hair growth in developing mice.

A new type of nerve cell involved in itch perception was discovered.

Overexpressing the epigen gene in mice leads to enlarged sebaceous glands and greasy fur.

Adenophora Radix extract can promote hair growth and increase melanin in mice.

African spiny mice can regenerate skin, hair, and cartilage, but not muscle, and their unique abilities could be useful for regenerative medicine.

The improved surgical method increases success and reduces fetal loss in fetal mouse models for scarless skin healing.

Removing both Atr and Trp53 genes in adult mice causes severe tissue damage and death due to DNA damage.

Male-to-female skin grafts in mice are rejected due to sex-linked antigens.

A gene mutation in mice causes hair loss, weak bones, and protein buildup, showing how protein processing issues can lead to diseases.

A position effect on the TRPS1 gene causes excessive hair growth in humans and mice.

miR-200a reduces goose fibroblast growth by targeting PITX2 in the Wnt pathway.

Serotype 4 of Ureaplasma showed the highest pathogenicity in female mice.

MRL/MpJ mice heal burns slower with more scarring and less tissue regeneration than BALB/c mice.

A gene deletion causes the "hairless" trait in Iffa Credo rats.

Overexpression of the HE4 gene in mice causes eye inflammation and cloudiness.

Methamphetamine affects gene expression in rat whisker follicles, with key genes linked to addiction.

The Naked gene in mice causes abnormal sebaceous glands and disrupts hair follicle organization.

The mouse model could be useful for baldness research and testing treatments like testosterone, cyproterone acetate, and minoxidil.

Tranexamic acid turns white hair brown in certain mice by affecting specific proteins.

Alopecia areata is influenced by genetics and immune system factors, and better understanding could improve treatments.

The angora mouse mutation causes long hair and hair defects due to a gene deletion.

The TRPV3 gene mutation affects hair growth by keeping mice in the growth phase longer, which could help treat hair loss.

rRSPO1 protein boosts hair growth by activating a key signaling pathway.

Altering the keratin 17 gene in mice hair follicles caused temporary hair issues, but changes were minimal and short-lived.

A new mouse model effectively mimics vitiligo for research and drug testing.