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The transcription factor Meis2 is essential for touch sensation and proper nerve development in touch receptors.

Activating the GDNF-GFRα1-RET signaling pathway could potentially promote skin and limb regeneration in humans and could be used to treat hair loss and promote wound healing.

β-estradiol slows hair growth in mice.

Serotype 4 of Ureaplasma showed the highest pathogenicity in female mice.

RNase L suppresses regeneration in mammals.

Finasteride reduces certain behaviors caused by D1-like receptor agonists but not by D2-like receptor agonists in mice.

Artificially inducing hair regrowth in mice can change the normal pattern and timing of hair growth, with minimal color differences between old and new fur.

miR-29 is a key factor that accelerates aging.

ANE syndrome is caused by a mutation in the RBM28 protein that disrupts ribosome assembly.

RORs may influence cashmere growth cycles.

Overexpressing the glucocorticoid receptor in mice leads to abnormal skin development and reduced inflammation.

Mutations in the hairless gene in mice affect its expression and lead to a range of developmental issues in multiple tissues.

The spiny mouse regenerates ear tissue asymmetrically, with gene expression differences possibly explaining its unique healing abilities.

TSC2-/meth cells can cause skin lesions, hair growth, and lung issues, and may be treated with chromatin remodeling agents.

The antibody 005-C04 blocks prolactin receptors, causing reversible infertility, impaired lactation, and hair regrowth in female mice.

Deleting the PTEN gene in mice causes nerve cells to grow larger and heal better after injury, but may cause overgrowth and hair loss in older mice.

Pygopus 2 helps ovarian cancer cells grow by aiding ribosomal RNA production, independent of Wnt signaling.

Sex steroids affect the MafB gene differently in male and female hamsters.

The gene Endothelin 3 makes mice's fur darker by increasing pigment cells and pigment levels.

Cells from a skin condition can create new hair follicles and similar growths in mice, and a specific treatment can reduce these effects.

Mice with a Gsdma3 gene mutation have thicker skin and longer hair follicle openings due to increased β-catenin levels.

Prdm1 is necessary for early whisker development in mice but not for other hair, and its absence changes nerve and brain patterns related to whiskers.

Losing both ERBB2 and ERBB3 receptors in mice causes significant skin problems and inflammation.

Different enzymes are active in different parts of developing mouse organs.

Rac1 is crucial for normal hair structure and pigmentation.

Certain mice without specific receptors or mast cells don't lose hair from stress.

Vaccines and targeting TrxR variants can help prevent cancer and reduce metastasis.

Lhx2 helps retinal cells respond to signals for eye development.