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A new gene mutation causes vitamin D resistance and rickets, treatable with calcium therapy.

GRHL3 is important for controlling gene activity in skin cells during different stages of their development.

Neuregulin3 affects cell development in the skin and mammary glands.

Revertant cell therapy shows promise for treating type XVII collagen deficiency, but better cell selection methods are needed.

Two siblings with a genetic mutation had a form of rickets that doesn't respond to vitamin D.

Some prostate cancers have gene changes that may affect treatment with certain drugs.

ETS2 drives cancer progression in squamous cell carcinoma and is linked to poor patient outcomes.

Hairless protein can block vitamin D activation in skin cells.

A20 protein is crucial for normal skin and hair development.

Two enzymes regulate androgen receptor activity, affecting treatments for androgen insufficiency and benign prostatic hyperplasia.

EDA signaling is linked to skin disorders, various cancers, and liver disease.

A mutation in the iRhom2 gene causes hairless mice due to abnormal hair follicle development.

Researchers found a gene in hamsters that responds to male hormones and may be indirectly controlled by them.

EGFR helps hair follicles transition properly by controlling Stathmin levels.

Drug repositioning offers hope for new, affordable treatments for a genetic skin disorder called ARCI.

Vaccines and targeting TrxR variants can help prevent cancer and reduce metastasis.

Lrig1 marks a unique group of stem cells in mouse skin that can become different skin cell types.

Human bone cells mainly produce a type 1 enzyme that may help regulate bone health.

The study concludes that the EDA2R gene is activated by p53 during chemotherapy but is not necessary for chemotherapy-induced hair loss.

The protein p53 directly reduces the production of Keratin 17, a skin and hair protein, in rats with radiation dermatitis.

FER and TOR signaling help root hair growth in low temperature and low nitrate conditions.

Two new enzymes, PAD-R11 and PAD-R4, were cloned and showed activity, with PAD-R11 resembling epidermal enzymes.

Low ERCC3 gene activity is linked to non-pigmented hair growth.

rRSPO1 protein boosts hair growth by activating a key signaling pathway.

The balance between cell renewal and differentiation controls the growth of cancerous cells in mouse skin.

Stem cells control their future role by changing ERK signal timing, affecting tissue regeneration and cancer.

Thioredoxin reductase 1 does not affect glucocorticoid receptor activity in hair follicle cells.

Erdr1 could be a new marker for diagnosing hair loss.

Krt6a-Cre transgenic mice help study gene effects on hair follicle development and tumor suppression.