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The vitamin D receptor has many roles in the body beyond managing calcium, affecting the immune system, hair growth, muscles, fat, bone marrow, and cancer cells.

A gene mutation causes curly hair and hair loss in rats.

Ribonucleotide excision repair is crucial to prevent skin cancer.

SETDB1 is essential for controlling DNA methylation, silencing retrotransposons, and maintaining skin cell health, with its absence leading to skin inflammation and hair loss.

Retinoic acid-binding proteins in skin are regulated by β-catenin and Notch signalling.

ERULUS controls root hair growth by regulating cell wall composition and pectin activity.

RXRa is crucial for Th2 immune cell development and may link nutrition to immune health.

Fibroblasts from healthy donors can prevent changes seen in recessive epidermolysis bullosa simplex.

Vitamin D receptor can control the hairless gene linked to hair loss even without vitamin D.

Non-coding RNA boosts retinoic acid production and signaling, aiding regeneration.

Vitamin D receptor and hairless protein are essential for hair growth.

Epiregulin can help hair grow and may be useful for treating hair loss.

Losing both ERBB2 and ERBB3 receptors in mice causes significant skin problems and inflammation.

ELL is crucial for gene transcription related to skin cell growth.

Retinoic acid receptors are important for hair follicle development.

Androgen receptors help prostate cancer cells grow and resist drugs.

Estrogen Receptor ß (ERß) is the main hormone controller in human skin and hair follicles, not Estrogen Receptor α (ERα) or the Androgen Receptor (AR).

RAR-gamma 1 is important for normal skin maintenance and differentiation.

Increasing Rps14 helps grow more inner ear cells and repair hearing cells in baby mice.

RXR agonists may promote hair growth in humans.

Certain mutations in the hairless protein disrupt its ability to regulate the hair cycle.

Retinoids increase steroid sulfatase activity in leukemia cells through RARα/RXR and involves certain pathways like phosphoinositide 3-kinase and ERK-MAP kinase.

Key genes can rewire networks, changing skin appendage types.

Different Hairless isoforms affect Vitamin D receptor activity in hair regulation, with one repressing and the other stimulating it.

RXRα is crucial for hair growth and skin cell function.

Researchers fixed gene mutations causing a skin disease in stem cells, which then improved skin grafts in mice.

A specific gene mutation causes vitamin D resistance, but certain vitamin D analogs might help.