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Activating cannabinoid receptor 1 reduces certain keratin levels, potentially aiding psoriasis treatment.

UVB exposure affects retinoid metabolism, which is important for skin cancer progression and treatment.

Krt15+ cells in mice can resist radiation, regenerate tissue, and start tumors, suggesting new cancer treatment targets.

Hair follicle cells and skin cells differ in keratinization and other features.

Myeloid cells can turn into skin and hair cells to help heal wounds.

Removing Ctip2 in skin cells causes skin inflammation similar to atopic dermatitis.

Sonic hedgehog signaling is needed for the development of touch-receptor cells in the skin, and the loss of Polycomb repressive complex 2 can lead to more of these cells.

Only one K16 gene on chromosome 17 makes a functional keratin protein.

A unique hair tumor with a rippled pattern was identified, showing incomplete differentiation and unusual cell arrangements.

Most U.S. keratinocyte carcinoma patients are older white males living in urban areas.

Loss of the p53 gene alone causes tumors, and losing both p53 and Rb genes speeds up aggressive skin cancer.

Alopecia in VDR knockout mice is due to a defect in hair cycle initiation, not keratinocyte issues.

Some cells may slow melanoma growth, a protein could affect skin pigmentation, a gene-silencing method might treat hair defects, skin bacteria changes likely result from eczema, and a defensin protein could help treat multiple sclerosis.

Keratinocytes contribute to hidradenitis suppurativa by causing inflammation and worsening the condition.

A rare skin reaction from cancer treatment was successfully managed with topical treatments and antihistamines.

Hairless protein can block vitamin D activation in skin cells.

CK 19 expression is higher in more severe skin cancers.

Keratin 17 is crucial for normal hair growth by regulating hair cycle transitions with TNFα.

Versican in dermal papilla cells is crucial for healthy hair growth.

Mutant stem cells adapt their metabolism differently to outcompete normal cells in the skin.

New findings suggest potential treatments for melanoma, hyperpigmentation, hair defects, and multiple sclerosis, and show skin microbiome changes don't cause atopic dermatitis.

Keratin 17 gene mutations cause both steatocystoma multiplex and pachyonychia congenita type 2.

Apoptotic cells may trigger cell death in hair follicles during their regression cycle.

Epithelial-derived Pop-Up Keratinocytes (ePUKs) may enhance wound healing in regenerative medicine.

Activating the hexosamine pathway can improve skin health and increase hair follicle stem cells.