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More research is needed to confirm sperm DNA fragmentation as a reliable tool for diagnosing male infertility.

Deleting the MAD2L1 gene in mice led to rapid tumor growth despite chromosomal instability.

Early detection of specific VDR mutations is crucial for effective treatment and better dental outcomes in children with hereditary vitamin D–resistant rickets.

Removing certain hair follicle stem cells worsens skin reactions to allergens.

Temporary ROS production in cultured human hair follicles promotes growth and stem cell activation.

DNAJB9 cfRNA could help diagnose and treat female hair loss.

Dermoscopy can help assess the severity of chronic radiation-induced skin damage in head and neck cancer patients.

Sulfur-containing radioprotectors can protect hair from X-ray damage if given before exposure but worsen damage if given after.

Disrupting Bcl-xL in mice reduces skin cancer risk.

Genetic mutations that disrupt homocysteine breakdown lead to increased damage in mouse hair keratin.

The L412F variant of TLR3 is linked to skin infections, more viral infections, and autoimmune issues.

A new mutation in the STING protein causes a range of symptoms and its severity may be affected by other genetic variations; treatment with a specific inhibitor showed improvement in one patient.

Reducing SFRP1 can promote hair growth and may help treat hair loss.

A 30 Gy radiation dose effectively causes severe radiodermatitis in rabbits.

Sfrp2 increases during hair follicle catagen phase and slows keratinocyte growth.

Activated protein C helps protect mice from long-term radiation damage.

Removing EGFR in skin causes inflammation and abnormal hair growth.

Boosting certain cell signals can prevent hair loss from chemotherapy and radiation.

A mutation in the KRT86 gene causes hair fragility in a Turkish family.

1319-nm laser radiation can cause skin damage, with severity depending on exposure time and beam size.

Heavy ion radiation has a more severe and long-lasting effect on mouse intestinal metabolites than gamma radiation.

EGFRi/MEKi treatments cause hair follicles to lose some immune protection, leading to inflammation.

TLR3 activation helps improve skin and hair follicle healing in mice.

Inhibiting the PI3K/Akt pathway may help prevent radiation-induced liver injury.

Plakophilin 1 helps control skin cell immune responses to prevent excessive inflammation.

Deleting Rac1 in the skin depletes stem cells and damages hair follicles.

Longer exposure to high-power millimeter wave radiation causes more severe skin damage in mice.

The Hr protein binds to DNA, interacts with p53, and affects cell cycle genes.

ROR2 is crucial for hair follicle stem cell maintenance and self-renewal.

Removing part of the vitamin D receptor stops vitamin D from working properly.