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AGD1 is important for root hair development in Arabidopsis, working with phosphoinositide signaling and the actin cytoskeleton.

The RHD4 gene is crucial for consistent root hair growth in Arabidopsis thaliana.

Phospholipids help plant proteins move by regulating receptor interactions.

A protein called GIS3 is important for the growth of root hairs in Arabidopsis by controlling two genes with the help of certain growth signals.

A girl had rickets due to a gene mutation affecting vitamin D response.

White lupin uses specific genes to grow root hairs and access phosphorus when it's scarce.

Bacillus amyloliquefaciens can boost root hair growth in some Arabidopsis plants, potentially improving agriculture.

A feedback loop between LRH and RSL4 controls root hair growth in Arabidopsis.

Patients with the same genetic mutation for vitamin D-resistant rickets showed different symptoms but all improved with treatment except for hair loss.

HLD10 can include increased body hair and Mongolian spots.

Three siblings with a genetic form of rickets showed different symptoms of the disease.

The enzyme RDH12 plays a role in vision and retinal disease, with mutations leading to early onset visual loss and blindness, but the exact disease mechanism is unclear and there are no treatments yet.

Rhupus is a complex syndrome that is hard to diagnose due to unclear clinical criteria.

RHUPUS should be considered in children with deforming arthritis.

Rhupus is a complex syndrome that combines rheumatoid arthritis and lupus, making diagnosis challenging.

HVDRR is caused by VDR gene mutations, leading to vitamin D resistance, treatable with high calcium doses, but alopecia remains permanent.

Hoyeraal-Hreidarsson syndrome in infants causes severe gastrointestinal issues.

A vitamin D receptor mutation causes rickets and affects immune responses.

Two different mutations in the vitamin D receptor gene cause different symptoms and responses to treatment in Lebanese patients with hereditary rickets.

A 72-year-old man was diagnosed with a rare skin form of Rosai-Dorfman disease after years of misdiagnosis.

A specific gene mutation causes sparse, brittle hair in a family.

A specific gene mutation causes vitamin D resistance, but certain vitamin D analogs might help.

New mutations in the VDR gene cause vitamin D-resistant rickets without hair loss.

Different harmful mutations in the CDH3 gene cause HJMD, but symptoms vary among individuals.

The condition is linked to chromosome 12, but no mutations were found in the known genes.

Two siblings with a genetic mutation had a form of rickets that doesn't respond to vitamin D.

A Chinese family had a child with a specific gene mutation causing vitamin D-resistant rickets, but the child improved with calcium and low-dose calcitriol.

Some people have genetic mutations that make them resistant to vitamin D, leading to rickets even with enough vitamin D intake.

VDDR I and II are genetic disorders affecting vitamin D use, causing rickets, with VDDR I treatable by vitamin D supplements and VDDR II needing high doses and calcium.

Rapp-Hodgkin syndrome, AEC, and EEC are different expressions of the same genetic disorder caused by TP63 gene mutations.