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Androgen receptor inactivation delays skin cancer onset and reduces incidence in mice, with gender differences in susceptibility.

The gene therapy showed significant wound healing and was safe for treating severe RDEB.

Blocking the mineralocorticoid receptor can help treat skin thinning caused by steroids.

Trichothiodystrophy hair is structurally abnormal with protein and organization issues.

Hair root analysis can effectively detect somatic mosaicism in double cortex syndrome.

Four-amino acid part makes enzyme sensitive to finasteride.

Thyrotropin-Releasing Hormone helps heal wounds in frog and human skin.

Targeting impaired Nrf2 signaling might help treat hidradenitis suppurativa early on.

Urtica dioica extract may help treat androgenic skin diseases by reducing a specific gene's activity.

The vitamin D receptor is essential for skin stem cells to grow, move, and become different cell types needed for skin healing.

A new genetic mutation linked to CARASIL syndrome and small artery disease was found in a Chinese family.

A new gene mutation causes female pseudohermaphroditism due to glucocorticoid resistance.

Most genetic mutations causing hypohidrotic ectodermal dysplasia in Russian patients are found in the EDA gene.

The protein folliculin, involved in a rare disease, works with another protein to control how cells stick together and their organization, and changes in this interaction can lead to disease symptoms.

A new gene mutation causes insulin resistance in a girl and her mother.

PRX01, PRX44, and PRX73 affect root hair growth by interacting with extensins in Arabidopsis.

Krt6a-Cre transgenic mice help study gene effects on hair follicle development and tumor suppression.

ODC expression in mouse skin and tumors is varied and can be inhibited by retinoic acid or cycloheximide.

A new mutation in the LIPH gene was found to cause a rare hair disorder in a Japanese boy.

FoxN1 overexpression in young mice harms immune cell and skin development.

A woman with acromegaloidism and normal growth hormone levels had a rare X-Tetrasomy, suggesting a need to study X-chromosome genes for their role in growth and facial development.

Key genes can rewire networks, changing skin appendage types.

A rare gene variant causes hair and nail issues in a family.

A rare genetic mutation causes resistance to vitamin D, leading to severe rickets and requiring high doses of calcium and vitamin D for treatment.

Planarians regenerate using conserved gene expression mechanisms, with runt-1 crucial for cell type specification.

Notch1 helps skin heal by attracting specific immune cells.

OX40-targeted therapies may help treat skin diseases by reducing inflammation and balancing immune responses.