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The 23rd Annual Scientific Meeting of the International Society of Hair Restoration Surgery in Chicago provided valuable information for daily practice in the field.

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Proper management of the dilation program is crucial after male-to-female reassignment surgery.

DA liposomes with chloramphenicol effectively target hair follicles and combat MRSA with minimal skin toxicity.

A new mutation in the TRPS1 gene caused Trichorhinophalangeal syndrome in a 17-year-old, highlighting the need for genetic testing.

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The S250C variant in a gene may cause autoimmunity and immunodeficiency by impairing protein function.

The serum improved hair growth and condition after hair transplant.

The mineralocorticoid receptor may play a role in skin and hair health and could be a new target for treating related disorders.

A new mutation in the mitochondrial DNA was found in a boy with MELAS, even though his family didn't show typical signs.

Mint water extract boosts protective enzymes in skin cells.

X-linked mental retardation includes various syndromes with both mental and physical abnormalities.

Four bacteria from the lab were found to be resistant to selenium.

Combining soursop leaf extract with doxorubicin may improve breast cancer treatment and reduce side effects.

Certain genetic variants impair enzyme activity, contributing to non-classic congenital adrenal hyperplasia.

A silicone treatment makes damaged hair more water-resistant and stronger.

MaasaLong may improve male health, but consult a doctor before use.

SARMs show promise but need more evidence to prove they're better than traditional androgens.

Overexpression of a specific receptor in mice skin causes skin thinning, early skin barrier formation, eye issues, and hair loss.

Genetic variants at 20p11 increase baldness risk in Chinese Han people.

Seborrheic dermatitis was most common in late adults and toddlers from 2016 to 2019.

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Male pattern baldness may increase COVID-19 severity risk.

miRNA-211 reduces melanin in Cashmere goats by suppressing AP1S2.

A person with a new mutation in the SCN1A gene developed brain inflammation after COVID-19.