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A gene deletion in mice causes weak protein, immune issues, hair loss, airway problems, and wasting disease.

The Gsdma3 gene is essential for normal hair development in mice.

Researchers created a new mouse model for studying Citrullinemia Type I and similar conditions, showing symptoms and treatment responses like those in humans.

Deleting part of a gene in mice causes wavy hair and high pup loss.

Introducing the rat OTC gene partially corrected OTC deficiency in mice.

The document concludes that careful design of genetic fate mapping experiments is crucial for accurate cell lineage tracing in mice.

Overexpressing Dsg3 in mice skin causes excessive cell growth and abnormal skin development.

Homozygous K5Cre transgenic mice have wavy hair and faster cancer progression.

Gene knockout mice developed scars similar to human hypertrophic scars, useful for studying scar progression.

Mice genetically modified to produce more CD109 in their skin had less inflammation and better healing with less scarring.

Researchers created a mouse model for Cushing's syndrome to study glucocorticoid excess and potential treatments.

Sox13 is a marker for early hair follicle development but not essential for skin and hair growth.

A new mouse model helps study melanocyte cells using GFP expression.

Cupric chloride treatment corrected abnormal Purkinje cell development in brindled mice.

Deleting the CD271 gene in mouse skin cells leads to disorganized skin and increased hair growth, suggesting CD271 is important for skin health.

EPI-NCSCs from hair follicles may help treat brain development issues in mice.

Mutations in claudin-1 and claudin-3 cause hair loss in baby mice.

The study created a mouse model to better understand hair follicle stem cells' role in hair growth and repair.

Stress can cause a type of hair loss in mice lacking the CCHCR1 gene.

The wavy coat in NCT mice is caused by multiple genes, including a mutation in the Prss53 gene.

Disrupting the Tsc2 gene in certain cells leads to thicker skin, larger hair, and changes in hair growth signaling, which can be partly reversed with specific treatment.

SCD1 is crucial for skin health and overall energy balance.

Tsc2-deficient stem cells can help understand and treat TSC-related tumors.

Mutations in the Sgk3 gene cause fuzzy hair in mice.

Syndecan-1 is essential for maintaining skin fat and preventing cold stress.

Krt6a-Cre transgenic mice help study gene effects on hair follicle development and tumor suppression.

A gene mutation in mice causes skin defects and early death.

The treatments stopped hair regrowth in mice.

Mice without certain skin enzymes have faster hair growth and bigger eye glands.