Search

everythinglearnresearchcommunity

for

Search:↓

Genetically modified stem cells from human hair follicles can lower blood sugar and increase survival in diabetic mice.

The study maps how genes are regulated during mouse hair growth.

Removing Gsdma1/2/3 genes reduces skin cell overgrowth by blocking a specific cell pathway.

Epidermal stem cells on a special membrane helped mice regrow full skin with hair and functions.

In vivo confocal scanning laser microscopy is an effective, non-invasive way to study and measure new hair growth after skin injury in mice.

Using a gene therapy with the Sonic Hedgehog gene helps mice regrow hair faster after losing it from chemotherapy.

MAA beads improved wound healing in male mice by activating the Shh pathway, but not in females.

Deleting TNFα gene reduces skin cancer risk in certain mice.

AIRE-deficient rats developed severe autoimmune disease similar to APECED, useful for testing treatments.

Inherited color dilution in Rex rabbits is linked to DNA methylation changes in hair follicles.

Polyamines are crucial for skin tumor development, and inhibiting them can prevent tumors.

The Foxn1(-/-) nude mouse shows disrupted and expanded skin stem cell areas due to high Lhx2 levels.

CRISPR-Cas9 can successfully edit genes in large mammals like Cashmere goats.

Kdm6b is crucial for skin cell differentiation.

Overexpression of SSAT in mice causes hair loss, liver damage, and sensitivity to polyamine analogues.

A new genetic disorder caused by an ODC1 mutation can be treated with DFMO.

Lgr5 and the vitamin D receptor are key in controlling skin inflammation and tumor risk in mice.

SETDB1 is essential for controlling DNA methylation, silencing retrotransposons, and maintaining skin cell health, with its absence leading to skin inflammation and hair loss.

SCUBE3 is a potential target for cancer and alopecia treatment but is challenging to target due to its varied roles.

Antiviral drugs, especially daclatasvir, may be a new treatment for a rare skin disease, improving survival and reducing symptoms in mice.

A female dog with mixed male and female traits was treated successfully with surgery.

A mutation in the Soat1 gene causes hair structure defects and other health issues in AKR/J mice.

Damaged hair follicles make mice more prone to skin inflammation and skin cancer after UV exposure.

New mutations in the SLC39A4 gene found in twins help understand the genetic cause of acrodermatitis enteropathica.

GFP transgenic mice help study cell origins in skin grafts.

Old C57BL/6 mice with unsynchronized hair cycles show less melanin in their spleens.

Mouse skin has special cells in the epidermis that decrease with age and are linked to keratinocyte stem cells.

Mice without the enzyme HSD17B3 still produce normal testosterone, suggesting they have different ways to make it compared to humans.

The ZIP13 variant is linked to abnormal hair quality.