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A new genetic tool improves the study of hair growth and potential hair disorder treatments.

Removing β-catenin in certain stem cells causes hair whitening and pigmentation issues.

Anti-Desmocollin 3 antibodies can cause atypical pemphigus symptoms.

The study created a mouse model to better understand hair follicle stem cells' role in hair growth and repair.

The curly mutation in SELH/Bc mice affects hair and may help study human genetic disorders.

A new mouse model helps study melanocyte cells using GFP expression.

The document concludes that careful design of genetic fate mapping experiments is crucial for accurate cell lineage tracing in mice.

A 4kb fragment of the desmocollin 3 promoter targets gene expression to specific skin and hair follicle areas.

The study created a mouse model that survives longer and shows fewer symptoms of pemphigus vulgaris.

A gene mutation in mice causes increased mast cells and disorganized hair follicles in their skin.

MC-DNA vector-based gene therapy can temporarily treat CBS deficiency in mice.

Mice with a Gsdma3 gene mutation have thicker skin and longer hair follicle openings due to increased β-catenin levels.

A gene deletion causes the "hairless" trait in Iffa Credo rats.

The HCR gene contributes to psoriasis risk.

Researchers created a new mouse model for studying Citrullinemia Type I and similar conditions, showing symptoms and treatment responses like those in humans.

The Gsdma3 gene is essential for normal hair development in mice.

The Megsin-Cre transgene is a new tool for genetic manipulation in the skin and upper digestive tract.

Homozygous K5Cre transgenic mice have wavy hair and faster cancer progression.

Deleting part of a gene in mice causes wavy hair and high pup loss.

A Gsdma3 mutation causes hair loss due to stem cell damage from skin inflammation.

Mutant CDP/Cux protein causes hair defects and reduced male fertility in mice.

The humanized AA mouse model is better for testing new alopecia areata treatments.

Scube3 gene affects mouse embryo growth in multiple areas, but needs more research.

Dsg1 is essential for maintaining a healthy skin barrier in mice.

A new mouse mutation causes skin and hair issues, influenced by another gene.

The G60S Connexin43 mutation causes hair growth issues and poor hair quality in mice, similar to human ODDD patients.

Mutations in the Sgk3 gene cause fuzzy hair in mice.

Researchers created a mouse with the same mutation as humans with trichothiodystrophy, showing similar symptoms and confirming the condition is due to defects in DNA repair and gene activity.

The treatments stopped hair regrowth in mice.