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IGF2BP3 and other m6A-related genes are linked to keloid formation and could be potential treatment targets.

Folliculotropic mycosis fungoides has unique molecular features and cell interactions that could guide targeted therapy.

A systems biology approach helps improve mesenchymal stromal cell therapies by mapping interactions and identifying treatment targets.

Understanding developmental pathways can improve wound healing treatments.

The scaffold improves wound healing and tissue regeneration.

Proinflammatory fibroblasts and vascular endothelial cells are key in keloid development.

A fungal enzyme was used to make compounds more soluble, aiding drug discovery and crop protection.

Understanding cellular interactions in VCA may lead to better treatments and reduce rejection.

CYLD deficiency in skin tumors disrupts hair follicle cell processes and protein secretion.

Secukinumab reduces immune activity in hidradenitis suppurativa skin.

Low fucosylation boosts stem cell growth in the eye.

Skin tumors with CYLD cutaneous syndrome show more NF-κB activity and less organized collagen.

The method successfully isolates hair follicle stem cells from mice for research.

Cancer-associated fibroblasts promote tumor growth in skin cancer.

Targeting the actin cytoskeleton could improve skin healing and reduce scarring.

Topical IKKα inhibitors may help prevent CCS tumours.

Small molecules can help turn skin cells into sweat gland-like cells for potential skin repair.

The study identified a key protein involved in producing underarm odor and found ways to inhibit it.

Mimicking fetal wound environments may enable scarless healing in adults.

Ceramide synthase 4 is essential for maintaining skin barrier health.

Researchers improved a method to study individual cells in newborn mouse skin and found a way to assess the severity of a skin condition in humans.

Mutations in the PADI3 gene may cause central centrifugal cicatricial alopecia in women of African ancestry.

AcD scaffolds improve tissue repair and regeneration by combining stem cells with a supportive matrix.

Scientists used stem cells to create a model of the skin disease Epidermolysis Bullosa simplex, which helped them understand its molecular mechanisms and could aid in finding treatments.

Hair growth-related cells need the enzyme SCD1 to help maintain the area that supports hair growth.

The protein FLCN is involved in cellular cleanup and is regulated by ULK1.

The lentiviral array can monitor and predict gene activity during stem cell differentiation.

Cis-UCA can form new compounds without enzymes, and sulphide donors reduce its UVB-induced toxicity in skin cells.

CRISPR/Cas9 and prime editing can potentially fix skin disorder genes safely and effectively.

Using a perfusion system and 3D spheroid culture improves the growth of corneal cell layers for tissue engineering.