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Removing β-catenin in certain stem cells causes hair whitening and pigmentation issues.

B6J mice live longer before 24 months, but B6N mice live longer after; both strains show weight gain, increased food and water intake, and health issues as they age.

A defective gene causes hair loss and taste insensitivity in BTBR mice.

Researchers improved a method to study individual cells in newborn mouse skin and found a way to assess the severity of a skin condition in humans.

VDC-1101 shows potential as a treatment for canine cutaneous T-cell lymphoma.

XEDAR deficiency prevents muscle degeneration in EDA-A2 transgenic mice.

Cats with abnormal hair had DSG4 gene changes causing hair problems.

Researchers created a new mouse model, G4, that mimics human PCOS symptoms and links the condition to a specific gene.

Mice with extra human KLK14 had hair and skin problems, including weaker cell bonds and inflammation, linked to Netherton syndrome.

Older mice healed wounds better but lost more weight and might have weaker immune systems afterward.

Rhino mice show significant meibomian gland changes, making them a potential model for studying gland disorders.

The Celsr1 gene is crucial for normal hair patterning in mice.

Granulocyte colony stimulating factor helps heal wounds without scars.

The frizzy mouse and hairless rat mutations are due to changes in the Prss8 gene.

Homozygous K5Cre transgenic mice have wavy hair and faster cancer progression.

Hairless USP mice have enlarged skin cysts as they age.

High levels of the protein Flightless I worsen ulcerative colitis symptoms in mice.

Copper supplements during pregnancy improve survival and development in mutant mice.

SOCS3 treatment can prevent hair loss by stopping harmful immune responses.

Anti-Desmocollin 3 antibodies can cause atypical pemphigus symptoms.

A mutation in the Sgkl gene causes defective hair growth in mice.

The study developed a successful mouse model to study skin infections, highlighting the importance of choosing the right fungal strains.

Deleting the MAD2L1 gene is tolerated in certain mouse cancer models.

SKH1 hairless mice have identifiable epidermal stem cells with specific markers.

Loss of Ten1 in mice causes telomere shortening and symptoms similar to human dyskeratosis congenita.

The research showed that CRISPR/Cas9 can fix mutations causing a skin disease in stem cells, which then improved skin grafts in mice, but more work on safety and efficiency is needed.

Chemotherapy reduces splenic melanin in mice.

The hairless mutant gene causes early hair loss and affects skin and thymus development in mice.

Mice that can regenerate tissue have cells that pause in the cell cycle, which is important for healing, similar to axolotls.