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A gene deletion in mice causes weak protein, immune issues, hair loss, airway problems, and wasting disease.

Mutations in the Sgk3 gene cause fuzzy hair in mice.

Using special RNA to target a mutant gene fixed hair problems in mice.

Scientists created a cell model to study and find treatments for a skin disease called RDEB.

Normal cells outcompete and remove mutant cells in the pancreas with the help of the EphA2 receptor.

FoxN1 overexpression in young mice harms immune cell and skin development.

PP2Acα is essential for proper hair and skin development.

Overexpression of the glucocorticoid receptor in mice causes developmental defects similar to ectodermal dysplasia.

Hoxc8 gene helps start mammary gland development by controlling specific signals.

Hair loss in certain mice is linked to changes in keratin-related genes.

Ectodysplasin-A1 is crucial for developing hair, teeth, and glands.

Overexpressing 14-3-3σ in mice skin reduces cell growth and hair density.

A mutation in mice causes hair loss and skin issues due to a defect in a gene affecting cell adhesion.

Tsc2-deficient stem cells can help understand and treat TSC-related tumors.

Blocking EGFR in mice causes hair loss and skin changes.

K16 can partially replace K14 but causes hair loss and skin issues.

The research cloned keratin 7 genes from humans, mice, and marsupials, found similarities between human and mouse genes, and discovered new areas of K7 expression in mice.

A new mouse model effectively mimics vitiligo for research and drug testing.

Vitamin D receptor knockout mice have significant motor impairments but no cognitive deficits.

Secreted inhibitors of Wnt and IGF signaling control hair and tooth development, creating species-specific patterns.

Scientists made a mouse that can be made to lose hair and then grow it back.

Disrupting Acvr1b in mice causes severe hair loss and thicker skin.

The DiLiCre mouse model is an effective tool for precise genome editing using light.

Li2CO3 improved skin disease in a mouse model of Focal Dermal Hypoplasia without toxicity.

Mice lacking the PPARγ gene in their fat cells had almost no fat tissue, severe metabolic problems, and abnormal development of other fat-related tissues.

The treatments stopped hair regrowth in mice.

Antizyme slows skin tumor growth by reducing cell growth in mice.

A new mutation in mice causes crooked whiskers and messy hair.

Activated erbB-2 in mice skin causes severe skin and hair abnormalities.