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The system allows precise control of gene expression in mouse skin, useful for studying skin biology.

Secreted inhibitors of Wnt and IGF signaling control hair and tooth development, creating species-specific patterns.

Scientists made a mouse that can be made to lose hair and then grow it back.

Fibroblasts from healthy donors can prevent changes seen in recessive epidermolysis bullosa simplex.

Activated erbB-2 in mice skin causes severe skin and hair abnormalities.

Mice lacking the PPARγ gene in their fat cells had almost no fat tissue, severe metabolic problems, and abnormal development of other fat-related tissues.

A specific gene segment can make mouse skin cells glow, helping study hair growth and gene effects.

Overexpressing 14-3-3σ in mice skin reduces cell growth and hair density.

PP2Acα is essential for proper hair and skin development.

Keratin 16 delays skin maturation and affects skin and hair development in mice.

Overexpression of the glucocorticoid receptor in mice causes developmental defects similar to ectodermal dysplasia.

Deleting Smad4 and PTEN genes in mice causes rapid, invasive stomach cancer.

Using special RNA to target a mutant gene fixed hair problems in mice.

A mutation in mice causes hair loss and skin issues due to a defect in a gene affecting cell adhesion.

Tmem50b and 2610305D13Rik genes play key roles in early mouse embryo development.

The naked mutation in mice causes hair loss and helps identify keratin genes.

CRISPR gene editing reduces harmful molecules in cells from Emery–Dreifuss Muscular Dystrophy patients.

Injecting specific cells into the skin can help improve skin structure and reduce blisters in a genetic skin disorder.

Noggin overexpression delays eyelid opening by affecting cell death and skin cell development.

Deleting Snai2 and Snai3 causes fatal autoimmunity.

A specific metabolite, not a receptor protein, triggers the production of certain kidney enzymes, but this process is disrupted in mice with a mutation causing testicular feminization.

Dkk4 is necessary for the initial development and arrangement of hair follicles.

Ectodysplasin inhibits Wnt signaling to help form hair follicles.

A mutation in the Adam10 gene causes freckle-like spots on Hairless mice.

Melanoma cells lose their ability to form tumors when placed in a zebrafish embryo environment.

Mice with the ab gene have abnormal sebaceous gland development, affecting sebum production.

Tabby mutations in mice affect hair follicle development and help study genetic mapping and certain medical conditions.