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Mice lacking Insig proteins had hair growth problems due to cholesterol buildup, but this was fixed by the drug simvastatin.

The SHJH hr mice with a mutated Hr gene show signs of faster skin aging due to poor antioxidative protection.

The scraggly mutation causes hair loss and skin defects in mice.

The Foxn1 gene is essential for normal nail and hair development.

The Dfl mutation in mice causes poor sebaceous gland function and complete hair loss.

Spiny mice are better at regenerating hair after injury than laboratory mice and could help us understand how to improve human skin repair.

Hairless mice have longer hair follicles and abnormal structures during the catagen phase.

Two mouse mutants have defective hair cuticle cross-linking.

Dsg1 is essential for maintaining a healthy skin barrier in mice.

African spiny mice can regenerate skin and hair after wounds due to specific tissue mechanics.

Mouse nails are similar to human nails, making them useful for studying nail diseases.

Naked-mouse hair lacks certain proteins and has less soluble fibril.

Proteomic analysis can identify genetic differences in mouse hair, helping understand hair defects and variations.

Mouse tail skin has different keratinization near hair follicles and scales.

The Naked (N) trait in mice is linked to lower glycine and tyrosine in hair proteins.

The Bsk mutation doesn't involve keratin gene recombination and its cause is unknown.

Injecting α-MSH made mice's hair turn black.

Spiny mice regenerate skin better than laboratory mice due to larger hair bulges, more stem cells, and different collagen ratios.

Abnormal keratin expression in mice causes severe oral issues, affecting feeding.

A litter of cats had a hair condition similar to a mouse mutation, leading to hair loss and abnormal hair and skin.

MRL/MpJ mice's skin wounds heal with scars, unlike their ear wounds which can regenerate.

The study created a mouse model that survives longer and shows fewer symptoms of pemphigus vulgaris.

MRL/MpJ mice heal burns slower with more scarring and less tissue regeneration than BALB/c mice.

The study created a mouse model to mimic degenerative diseases for testing tissue repair and new therapies.

Mitochondrial problems in tooth cells lead to bad enamel and dentin development in mice.

A mutation in the Adam10 gene causes freckle-like spots on Hairless mice.

MAA beads improved wound healing in male mice by activating the Shh pathway, but not in females.

Damaged hair follicles make mice more prone to skin inflammation and skin cancer after UV exposure.

The harlequin ichthyosis mouse mutation causes thick skin and early death, resembling a human skin disorder.

Mutant MK6a transgenes in mice cause blistering, hair loss, and potential human alopecia.