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Sonic hedgehog signaling is needed for the development of touch-receptor cells in the skin, and the loss of Polycomb repressive complex 2 can lead to more of these cells.

γδ T cells adapt uniquely to different tissues in mice.

The Celsr1 gene is crucial for normal hair patterning in mice.

The study concluded that hair growth in mice is regulated by a stable interaction between skin cell types, and disrupting this can cause hair loss.

Activating Kras in mouse skin causes excess skin and hair loss.

Zfp157 is active in many mouse tissues during development and in specific adult cells.

Blocking JAK-STAT5 signaling in mice leads to hair growth.

Topical estrogen treatments did not change hair growth in certain mouse strains, questioning previous findings on their role in hair growth control.

A new mutation in the HR gene is linked to a rare form of hair loss with limb deformities.

Radiation significantly slows down wound healing in mice.

Understanding the mouse hair cycle is crucial for cancer research.

M-CSF-stimulated myeloid cells can cause alopecia areata in mice.

Only Deomyinae rodents can regenerate complex tissues.

Finasteride reduces certain behaviors caused by D1-like receptor agonists but not by D2-like receptor agonists in mice.

The mutated hairless gene causes hair loss by acting as a new type of corepressor affecting thyroid hormone receptors.

Exosomes from small-tailed Han sheep enhance hair growth in mice.

Matriptase-2 helps control iron levels by suppressing hepcidin, and its deficiency can cause iron-deficiency anemia.

Over-expression of Sp2 can lead to cancer by preventing proper stem cell differentiation.

Retinoic acid affects male and female muscle energy use and function differently.

CDP is crucial for lung and hair follicle cell development.

Overexpressing NSD3 in mice causes breast cancer-like tumors and gland abnormalities.

Transplanted bone marrow cells actively move, form clusters, and grow after transplantation.

Hair growth and pigmentation in mice involve specific stages crucial for research.

Mouse and human keratin 16 can both form filaments, with differences likely due to the tail domain, not the helical domain.

The TRPV3 gene mutation affects hair growth by keeping mice in the growth phase longer, which could help treat hair loss.