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The fuzzy gene is crucial for controlling hair growth cycles.

Amino acid storage proteins are essential for maintaining stem cells in female fruit flies.

Exposure to finasteride in the womb caused lasting reproductive issues in male rats.

Finasteride exposure affects gene expression and anogenital distance in male rat fetuses.

The MAPK/ERK pathway is crucial in controlling cell growth and death in finasteride-induced hypospadias.

Ets2 gene is crucial for placental development in mice.

Mice without the enzyme HSD17B3 still produce normal testosterone, suggesting they have different ways to make it compared to humans.

Finasteride for hair loss may cause long-lasting sexual side effects in young males.

Older parental age, rural living, and specific genetic mutations increase hypospadias risk in children.

Finasteride can reliably cause hypospadias in rabbits.

Finasteride, a hair loss drug, has unaddressed sexual side effects due to the manufacturer's inadequate safety measures and lack of additional studies.

Some common medications may harm male fertility by affecting hormones, sperm production, and sexual function.

Hesperidin helps protect against testicular damage caused by finasteride.

Dutasteride does not seem to change rat spermatogenesis or oxidative stress.

DA-9401 can protect against finasteride-induced reproductive damage in rats.

Some drugs can reduce male semen quality.

Testosterone increases acidification in the vas deferens by up-regulating V-ATPase.

Genetic mutations can affect female sexual development, requiring personalized medical care.

A new method for treating male pattern baldness in men with a low chance of improvement was discussed.

Atrazine causes birth defects in rats, and finasteride can reliably create a hypospadias model.

Finasteride may cause lasting sexual side effects, but more research is needed.

SSAT is a key enzyme affecting cell growth and metabolism, with potential but risky use in disease treatment.

Spermidine is essential for plant growth and adaptation to stress.

The Dfl mutation in mice causes poor sebaceous gland function and complete hair loss.

5-alpha reductase deficiency leads to male sexual development issues and treatments like finasteride help with prostate enlargement and hair loss.

The study found potential new DNA patterns in fertility genes, but further testing is needed.

Spermidine injections increased wool growth rate in lambs without changing fibre thickness.

Disrupting Smad4 in mouse skin causes early hair follicle stem cell activity that leads to their eventual depletion.

The study found that aging hair follicle stem cells change behavior, leading to hair loss, but drugs targeting these cells may help restore hair growth.