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Prostaglandin D2 increases testosterone levels in skin cells through reactive oxygen species, not enzymes, which could lead to new hair loss treatments.

The equine epididymis mainly uses type 1 5α-reductase, and both finasteride and dutasteride can inhibit its activity.

Curcumin and its derivatives can block an enzyme important for making testosterone, with one derivative being particularly strong.

DHEA affects various tissues by interacting with multiple receptors and can be converted into sex hormones.

Researchers found a gene in hamsters that responds to male hormones and may be indirectly controlled by them.

Androgens affect skin conditions like acne and hair loss through specific biochemical pathways.

Prostaglandin D2 increases testosterone production in skin cells through a process involving reactive oxygen species, which could be a new target for treating hair loss and other skin conditions driven by testosterone.

Androgens and estrogens are crucial for insulin secretion in males.

Cortisol and corticosterone thin mouse skin and regress hair follicles, while testosterone promotes skin and hair development.

The enzyme system in hair follicles is similar to the liver's and is affected by certain inhibitors.

Scientists found new nonsteroidal compounds that can act like natural male hormones and might help treat male fertility and hormone issues.

New chemical compounds were made that effectively block an enzyme linked to prostate growth.

Vitamin D and glucocorticoids affect bone growth, metabolism, and immune response.

Androgenic steroids can slow down or stop the growth of certain skin fungi.

Selective non-steroidal inhibitors of 5α-reductase type 1 can help treat DHT-related disorders.

Bolandiol, a synthetic steroid, builds muscle and bone without greatly affecting sex glands, and works differently from other hormones.

Eicosanoids are crucial for skin health, and targeting their pathways may help treat skin conditions.

Blocking the enzyme 11β-HSD1 might help treat obesity and metabolic issues.

Beta-sitosterol and stigmasterol might help prevent hair loss by blocking a specific enzyme.

Steroid sulfatase in hair follicles may be a target for treating hair loss.

3-tetrazolo steroidal analogs can strongly inhibit the enzyme linked to hair loss.

Genetic variations greatly affect individual metabolism and can impact health and disease risk.

Smaller substituents at C-17 enhance the inhibitory activity of progesterone derivatives on 5alpha-reductase.

Androgens regulate Sertoli cells and affect fertility through specific receptors and coregulators.

Enzyme activities do not cause early pubic hair in these girls.

Increased 11β-HSD1 activity in skin may contribute to aging and could be targeted to reduce aging effects.

Glucocorticoid receptors help regulate genes important for skin health and hair growth.

Dexamethasone may influence hair growth by altering estrogen receptor activity in hair cells.