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Lef1 helps adult skin cells maintain their ability to heal wounds and regenerate hair, but the study's methods and conclusions have been questioned.

A new technology showed that the SOX9 gene might control heart scar formation after injury, suggesting new treatment possibilities.

A new mutation in the STING protein causes a range of symptoms and its severity may be affected by other genetic variations; treatment with a specific inhibitor showed improvement in one patient.

Piezo1 helps skin grow by managing metabolism and inflammation.

A new mutation in the TMEM173 gene and a risk allele in IFIH1 cause a unique set of immune-related symptoms.

The treatments stopped hair regrowth in mice.

Stem cells in hair follicles can become neurons and other cells, especially in the upper part, useful for nerve repair.

Multiple mouse desmoglein 1 isoforms have distinct roles in skin and hair development.

Csdc2 helps hair growth in cashmere goats by regulating specific genes.

CTHRC1 helps sweat glands recover by rebuilding nearby blood vessels.

MOF controls skin development by regulating genes for mitochondria and cilia.

Mouse zigzag hair bends form due to a 3-day cycle of changes in hair progenitors and their environment.

Double-stranded RNA activates a pathway that causes a skin protein to be expressed in the wrong place.

Zfp157 is active in many mouse tissues during development and in specific adult cells.

Chronic stress in mice changes skin metabolism and gene expression, leading to hair loss.

The method helps estimate and track skin cell growth and movement during healing.

Human hair follicles contain a complex network of prostanoid receptors that may influence hair growth.

The study created a mouse model to mimic degenerative diseases for testing tissue repair and new therapies.

Twist2 is essential for scarless skin healing and hair growth in mouse fetuses.

Blocking the HEDGEHOG-GLI1 pathway can reduce keloid growth and may be a potential treatment.

Targeting the HEDGEHOG-GLI1 pathway could help treat keloids.

A gene mutation in mice causes increased mast cells and disorganized hair follicles in their skin.

Keeping human skin stem cells is easier with low temperatures and mTOR inhibition.

PKC ε increases hair follicle stem cell turnover and may raise skin cancer risk.

Gab1 protein is crucial for hair growth and stem cell renewal, and Mapk signaling helps maintain these processes.

ARHGEF3 is essential for proper hair follicle development in mice.

Mice with extra sheep genes had hair that fell out and regrew in cycles.

The FOXN1 gene is crucial for developing immune cells and preventing immune disorders.

Porcine hair follicles are useful for studying keratinocyte function, with galectin-1 as a potential stem cell marker.